Protective effect of glutathione in HIV-1 lytic peptide 1-induced cell death in human neuronal cells (Q34093320)

31 July 2017

title

Protective effect of glutathione in HIV-1 lytic peptide 1-induced cell death in human neuronal cells (English)

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31 July 2017

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Sung JH

1

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31 July 2017

Shin SA

2

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31 July 2017

Park HK

3

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31 July 2017

Montelaro RC

4

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31 July 2017

Chong YH

5

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31 July 2017

publication date

1 October 2001

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31 July 2017

published in

Journal of NeuroVirology

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31 July 2017

volume

7

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31 July 2017

issue

5

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31 July 2017

page(s)

454-465

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Glutathione deficiency and human immunodeficiency virus infection

31 July 2017

cites work

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Glutathione deficiency is associated with impaired survival in HIV disease

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Both necrosis and apoptosis contribute to HIV-1-induced killing of CD4 cells

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HIV-1 Tat potentiates TNF-induced NF-kappa B activation and cytotoxicity by altering the cellular redox state

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Membrane alterations linked to early interactions of HIV with the cell surface

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HIV-1 modulates the expression of gelatinase A and B in monocytic cells

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Diminished glutathione levels cause spontaneous and mitochondria-mediated cell death in neurons from trisomy 16 mice: a model of Down's syndrome

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Mitochondrial control of cell death

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The mitochondrial death/life regulator in apoptosis and necrosis

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Characterization of hydrogen peroxide toxicity in cultured rat forebrain neurons.

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A synthetic peptide corresponding to the carboxy terminus of human immunodeficiency virus type 1 transmembrane glycoprotein induces alterations in the ionic permeability of Xenopus laevis oocytes.

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Mitochondrial dysfunctions in circulating T lymphocytes from human immunodeficiency virus-1 carriers

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10.1080/135502801753170318

Identifiers

DOI

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Dimensions Publication ID

31 July 2017

1031746719

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