The BRCA1-interacting protein Abraxas is required for genomic stability and tumor suppression. (Q34109150)

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10 March 2020

title

The BRCA1-interacting protein Abraxas is required for genomic stability and tumor suppression (English)

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10 March 2020

2

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10 March 2020

Ting-Hsiang Huang

4

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10 March 2020

Jessica Tyler

7

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10 March 2020

author name string

Andy Castillo

1

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10 March 2020

Baohua Sun

3

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10 March 2020

Yucai Wang

5

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10 March 2020

Stephanie A Yazinski

6

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10 March 2020

Lei Li

8

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10 March 2020

M James You

9

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10 March 2020

Lee Zou

10

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10 March 2020

Jun Yao

11

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10 March 2020

Bin Wang

12

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10 March 2020

publication date

24 July 2014

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10 March 2020

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10 March 2020

volume

8

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10 March 2020

issue

3

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10 March 2020

page(s)

807-817

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Cumulative haploinsufficiency and triplosensitivity drive aneuploidy patterns and shape the cancer genome

10 March 2020

cites work

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5 July 2018

Cancer genome landscapes

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Mouse models of BRCA1 and their application to breast cancer research

5 July 2018

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A distinct replication fork protection pathway connects Fanconi anemia tumor suppressors to RAD51-BRCA1/2.

5 July 2018

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BRCA1 functions independently of homologous recombination in DNA interstrand crosslink repair

5 July 2018

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BRCA1 tumor suppressor network: focusing on its tail

5 July 2018

1 reference

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Breast cancer-associated Abraxas mutation disrupts nuclear localization and DNA damage response functions

5 July 2018

1 reference

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BRCA1 tumor suppression depends on BRCT phosphoprotein binding, but not its E3 ligase activity

5 July 2018

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RAP80-directed tuning of BRCA1 homologous recombination function at ionizing radiation-induced nuclear foci

5 July 2018

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The BRCA1-RAP80 complex regulates DNA repair mechanism utilization by restricting end resection

5 July 2018

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Expanded roles of the Fanconi anemia pathway in preserving genomic stability

5 July 2018

1 reference

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Differential regulation of JAMM domain deubiquitinating enzyme activity within the RAP80 complex

5 July 2018

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COSMIC (the Catalogue of Somatic Mutations in Cancer): a resource to investigate acquired mutations in human cancer

5 July 2018

1 reference

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Recruitment of fanconi anemia and breast cancer proteins to DNA damage sites is differentially governed by replication

5 July 2018

1 reference

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NBA1, a new player in the Brca1 A complex, is required for DNA damage resistance and checkpoint control

5 July 2018

1 reference

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Cancer proliferation gene discovery through functional genomics.

5 July 2018

1 reference

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Ubc13/Rnf8 ubiquitin ligases control foci formation of the Rap80/Abraxas/Brca1/Brcc36 complex in response to DNA damage

5 July 2018

1 reference

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Emergence of a DNA-damage response network consisting of Fanconi anaemia and BRCA proteins

5 July 2018

1 reference

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CCDC98 is a BRCA1-BRCT domain-binding protein involved in the DNA damage response

5 July 2018

1 reference

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CCDC98 targets BRCA1 to DNA damage sites

5 July 2018

1 reference

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Abraxas and RAP80 form a BRCA1 protein complex required for the DNA damage response

5 July 2018

1 reference

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Insights into the molecular basis of human hereditary breast cancer from studies of the BRCA1 BRCT domain

5 July 2018

1 reference

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Multifactorial contributions to an acute DNA damage response by BRCA1/BARD1-containing complexes

5 July 2018

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Structural basis of phosphopeptide recognition by the BRCT domain of BRCA1.

5 July 2018

1 reference

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Structure of the BRCT Repeats of BRCA1 Bound to a BACH1 Phosphopeptide

5 July 2018

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Phosphopeptide Binding Specificities of BRCA1 COOH-terminal (BRCT) Domains

5 July 2018

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The BRCT Domain Is a Phospho-Protein Binding Domain

5 July 2018

1 reference

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BRCT Repeats As Phosphopeptide-Binding Modules Involved in Protein Targeting

5 July 2018

1 reference

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BRCA1-independent ubiquitination of FANCD2

5 July 2018

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Interaction of the Fanconi anemia proteins and BRCA1 in a common pathway

5 July 2018

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A high-throughput functional complementation assay for classification of BRCA1 missense variants

5 July 2018

1 reference

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Structure of the BRCT repeats of BRCA1 bound to a BACH1 phosphopeptide: implications for signaling

28 October 2018

1 reference

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20 years studying p53 functions in genetically engineered mice

28 October 2018

1 reference

PubMed

https://pubmed.ncbi.nlm.nih.gov/25066119

inferred from PubMed ID database lookup

12 December 2020

based on heuristic

Identifiers

DOI

10.1016/J.CELREP.2014.06.050

1 reference

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10 March 2020

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10 March 2020

PubMed publication ID

25066119

1 reference

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