Bmi1 is essential for cerebellar development and is overexpressed in human medulloblastomas (Q34306873)

1 August 2017

title

Bmi1 is essential for cerebellar development and is overexpressed in human medulloblastomas (English)

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1 August 2017

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Carly Leung

1

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1 August 2017

Merel Lingbeek

2

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1 August 2017

Olga Shakhova

3

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1 August 2017

James Liu

4

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1 August 2017

Ellen Tanger

5

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1 August 2017

Parvin Saremaslani

6

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1 August 2017

Maarten Van Lohuizen

7

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1 August 2017

Silvia Marino

8

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1 August 2017

language of work or name

English

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publication date

1 March 2004

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1 August 2017

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1 August 2017

volume

428

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1 August 2017

issue

6980

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1 August 2017

page(s)

337-341

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The oncogene and Polycomb-group gene bmi-1 regulates cell proliferation and senescence through the ink4a locus

1 August 2017

cites work

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Multiple docking sites on substrate proteins form a modular system that mediates recognition by ERK MAP kinase

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Posterior transformation, neurological abnormalities, and severe hematopoietic defects in mice with a targeted deletion of the bmi-1 proto-oncogene

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Nmyc upregulation by sonic hedgehog signaling promotes proliferation in developing cerebellar granule neuron precursors

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N-myc is essential during neurogenesis for the rapid expansion of progenitor cell populations and the inhibition of neuronal differentiation

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7 January 2021

Bmi-1 is required for maintenance of adult self-renewing haematopoietic stem cells

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7 January 2021

Bmi-1 determines the proliferative capacity of normal and leukaemic stem cells

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7 January 2021

Bmi-1 dependence distinguishes neural stem cell self-renewal from progenitor proliferation

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7 January 2021

Control of neuronal precursor proliferation in the cerebellum by Sonic Hedgehog

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Medulloblastoma: a problem of developmental biology

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Analysis of PTCH/SMO/SHH pathway genes in medulloblastoma.

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Mutations in SUFU predispose to medulloblastoma

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Identification of PATCHED mutations in medulloblastomas by direct sequencing

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Prediction of central nervous system embryonal tumour outcome based on gene expression

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Aggressive medulloblastoma with high-level N-myc amplification.

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Bmi-1 Regulation of INK4A-ARF Is a Downstream Requirement for Transformation of Hematopoietic Progenitors by E2a-Pbx1

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The zinc finger protein GLI transforms primary cells in cooperation with adenovirus E1A

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Development of a self-inactivating lentivirus vector.

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Identifiers

DOI

10.1038/NATURE02385

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Dimensions Publication ID

1 August 2017

1008729213

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