Microglial chemotaxis, activation, and phagocytosis of amyloid beta-peptide as linked phenomena in Alzheimer's disease (Q34386095)

1 August 2017

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title

Microglial chemotaxis, activation, and phagocytosis of amyloid beta-peptide as linked phenomena in Alzheimer's disease (English)

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1 August 2017

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Rogers J

1

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1 August 2017

Lue LF

series ordinal

2

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1 August 2017

publication date

1 November 2001

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Neurochemistry International

1 August 2017

published in

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1 August 2017

volume

39

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1 August 2017

issue

5-6

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1 August 2017

page(s)

333-340

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Granules in glial cells of patients with Alzheimer's disease are immunopositive for C-terminal sequences of beta-amyloid protein

1 August 2017

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

Fibrillar beta-amyloid induces microglial phagocytosis, expression of inducible nitric oxide synthase, and loss of a select population of neurons in the rat CNS in vivo.

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7 January 2021

Chemokines/chemokine receptors in the central nervous system and Alzheimer's disease

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7 January 2021

Amyloid-beta peptide-receptor for advanced glycation endproduct interaction elicits neuronal expression of macrophage-colony stimulating factor: a proinflammatory pathway in Alzheimer disease

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7 January 2021

Amyloid beta and amylin fibrils induce increases in proinflammatory cytokine and chemokine production by THP-1 cells and murine microglia

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7 January 2021

Inducible nitric oxide synthase expression is selectively induced in astrocytes isolated from adult human brain

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7 January 2021

10.1016/S0197-0186(01)00040-7

Identifiers

DOI

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1 August 2017

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