Matrix metalloproteinase expression in an experimentally-induced DTH model of multiple sclerosis in the rat CNS. (Q36897642)

16 August 2017

title

Matrix metalloproteinase expression in an experimentally-induced DTH model of multiple sclerosis in the rat CNS. (English)

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16 August 2017

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Anthony DC

1

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16 August 2017

Miller KM

2

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16 August 2017

Fearn S

3

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16 August 2017

Townsend MJ

4

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16 August 2017

Opdenakker G

5

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16 August 2017

Wells GM

6

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16 August 2017

Clements JM

7

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16 August 2017

Chandler S

8

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16 August 2017

Gearing AJ

9

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16 August 2017

Perry VH

10

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16 August 2017

publication date

1 July 1998

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Journal of Neuroimmunology

16 August 2017

published in

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16 August 2017

volume

87

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16 August 2017

issue

1-2

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16 August 2017

page(s)

62-72

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https://api.crossref.org/works/10.1016%2FS0165-5728%2898%2900046-0

16 August 2017

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Purification of rabbit bone inhibitor of collagenase

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Matrix metalloproteinases degrade myelin basic protein

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Matrix metalloproteinase expression during experimental autoimmune encephalomyelitis and effects of a combined matrix metalloproteinase and tumour necrosis factor-alpha inhibitor

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The Expression of Tissue-type Plasminogen Activator, Matrix Metalloproteases and Endogenous Inhibitors in the Central Nervous System in Multiple Sclerosis

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The roles of host and donor cells in the rejection of skin allografts by T cell-deprived rats injected with syngeneic T cells

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Processing of tumour necrosis factor-α precursor by metalloproteinases

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Matrix metalloproteinases and processing of pro-TNF-alpha.

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Reversal of experimental autoimmune encephalomyelitis with a hydroxamate inhibitor of matrix metalloproteases

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Matrilysin is expressed by lipid-laden macrophages at sites of potential rupture in atherosclerotic lesions and localizes to areas of versican deposition, a proteoglycan substrate for the enzyme

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Suppression of experimental allergic encephalomyelitis in the Lewis rat by the matrix metalloproteinase inhibitor Ro31-9790

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Heterogeneity in the distribution and morphology of microglia in the normal adult mouse brain

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Interferon beta-1b inhibits gelatinase secretion and in vitro migration of human T cells: a possible mechanism for treatment efficacy in multiple sclerosis

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Matrix metalloproteinases in the normal human central nervous system, microglial nodules, and multiple sclerosis lesions

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The matrix-degrading metalloproteinases

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Demyelination in the central nervous system following a delayed-type hypersensitivity response to bacillus Calmette-Guérin

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Delayed-type hypersensitivity lesions in the central nervous system are prevented by inhibitors of matrix metalloproteinases

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Ultrastructural studies of an immune-mediated inflammatory response in the CNS parenchyma directed against a non-CNS antigen

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Stromelysin is an activator of procollagenase. A study with natural and recombinant enzymes

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Matrix metalloproteinase degradation of elastin, type IV collagen and proteoglycan. A quantitative comparison of the activities of 95 kDa and 72 kDa gelatinases, stromelysins-1 and -2 and punctuated metalloproteinase (PUMP).

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Evidence for dendritic competition in the developing retina

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Leukocyte Gelatinase B Cleavage Releases Encephalitogens from Human Myelin Basic Protein

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The induction of 72-kD gelatinase in T cells upon adhesion to endothelial cells is VCAM-1 dependent

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Matrix metalloproteinases in brain injury

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Tumor necrosis factor-alpha-induced gelatinase B causes delayed opening of the blood-brain barrier: an expanded therapeutic window

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Brain extracellular matrix

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Characterization of MRI response to treatment with interferon beta-1b: contrast-enhancing MRI lesion frequency as a primary outcome measure

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The purification of tissue inhibitor of metalloproteinases-2 from its 72 kDa progelatinase complex. Demonstration of the biochemical similarities of tissue inhibitor of metalloproteinases-2 and tissue inhibitor of metalloproteinases-1.

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Quantitation of matrix metalloproteinases in cultured rat astrocytes using the polymerase chain reaction with a multi-competitor cDNA standard.

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Identifiers

DOI

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16 August 2017

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