Diabetic KKAy mice exhibit increased hepatic PPARgamma1 gene expression and develop hepatic steatosis upon chronic treatment with antidiabetic thiazolidinediones (Q38297979)

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scientific article published on July 2001

Language	Label	Description	Also known as
English	Diabetic KKAy mice exhibit increased hepatic PPARgamma1 gene expression and develop hepatic steatosis upon chronic treatment with antidiabetic thiazolidinediones	scientific article published on July 2001

Statements

scholarly article

1 reference

Europe PubMed Central

PubMed publication ID

1 September 2017

Diabetic KKAy mice exhibit increased hepatic PPARgamma1 gene expression and develop hepatic steatosis upon chronic treatment with antidiabetic thiazolidinediones (English)

1 reference

Europe PubMed Central

PubMed publication ID

1 September 2017

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based on heuristic

inferred from title

author name string

Bedoucha M

1

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Europe PubMed Central

PubMed publication ID

1 September 2017

Atzpodien E

2

1 reference

Europe PubMed Central

PubMed publication ID

1 September 2017

Boelsterli UA

3

1 reference

Europe PubMed Central

PubMed publication ID

1 September 2017

publication date

1 July 2001

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Europe PubMed Central

PubMed publication ID

1 September 2017

Journal of Hepatology

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Europe PubMed Central

PubMed publication ID

1 September 2017

35

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Europe PubMed Central

PubMed publication ID

1 September 2017

1

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Europe PubMed Central

PubMed publication ID

1 September 2017

17-23

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Europe PubMed Central

PubMed publication ID

1 September 2017

An antidiabetic thiazolidinedione is a high affinity ligand for peroxisome proliferator-activated receptor gamma (PPAR gamma)

1 reference

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Peroxisome proliferator-activated receptors: a nuclear receptor signaling pathway in lipid physiology

1 reference

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7 January 2021

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Regulation of Lipoprotein Metabolism by Thiazolidinediones Occurs through a Distinct but Complementary Mechanism Relative to Fibrates

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PPARγ Is Required for the Differentiation of Adipose Tissue In Vivo and In Vitro

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Peroxisome proliferator-activated receptor alpha-isoform deficiency leads to progressive dyslipidemia with sexually dimorphic obesity and steatosis

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Tissue distribution and quantification of the expression of mRNAs of peroxisome proliferator-activated receptors and liver X receptor-alpha in humans: no alteration in adipose tissue of obese and NIDDM patients

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Peroxisome proliferator-activated receptor alpha is restricted to hepatic parenchymal cells, not Kupffer cells: implications for the mechanism of action of peroxisome proliferators in hepatocarcinogenesis.

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Regulation of PPAR gamma gene expression by nutrition and obesity in rodents.

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General Survey of Diabetic Features of Yellow KK Mice

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Suppression of hepatic gluconeogenesis in long-term Troglitazone treated diabetic KK and C57BL/KsJ-db/db mice

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Troglitazone action is independent of adipose tissue

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Down-regulation by troglitazone of hepatic tumor necrosis factor-alpha and interleukin-6 mRNA expression in a murine model of non-insulin-dependent diabetes

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Quantitative real-time PCR for the measurement of feline cytokine mRNA.

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The nuclear eicosanoid receptor, PPARgamma, is aberrantly expressed in colonic cancers

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Effect of troglitazone (CS-045) and bezafibrate on glucose tolerance, liver glycogen synthase activity, and beta-oxidation in fructose-fed rats

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Tissue-specific actions of antidiabetic thiazolidinediones on the reduced fatty acid oxidation in skeletal muscle and liver of Zucker diabetic fatty rats.

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Differential activation of peroxisome proliferator-activated receptor-gamma by troglitazone and rosiglitazone

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Terminal differentiation of human breast cancer through PPAR gamma

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The insulin sensitizer, BRL 49653, reduces systemic fatty acid supply and utilization and tissue lipid availability in the rat.

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Pioglitazone: In vitro Effects on Rat Hepatoma Cells and in vivo Liver Hypertrophy in KKAv Mice

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Steatohepatitis: a tale of two "hits"?

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Fibrosis in chronic hepatitis C correlates significantly with body mass index and steatosis

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Identifiers

10.1016/S0168-8278(01)00066-6

1 reference

Europe PubMed Central

PubMed publication ID

1 September 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

1 September 2017

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