Upregulation of miR-21 by Ras in vivo and its role in tumor growth. (Q39642600)

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15 February 2020

title

Upregulation of miR-21 by Ras in vivo and its role in tumor growth (English)

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15 February 2020

9

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15 February 2020

Gabriella De Vita

14

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15 February 2020

Pietro Zoppoli

3

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15 February 2020

Carmen Guerra

4

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15 February 2020

Angelo Ferraro

5

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15 February 2020

Anna Maria Bello

6

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15 February 2020

Pasquale De Luca

7

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15 February 2020

Cecilia Calabrese

8

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15 February 2020

Michele Ceccarelli

10

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15 February 2020

author name string

D Frezzetti

1

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15 February 2020

M De Menna

2

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15 February 2020

M Zollo

11

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15 February 2020

M Barbacid

12

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15 February 2020

R Di Lauro

13

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language of work or name

English

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publication date

18 October 2010

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15 February 2020

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15 February 2020

volume

30

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15 February 2020

issue

3

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15 February 2020

page(s)

275-286

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Ras induces chromosome instability and abrogation of the DNA damage response.

15 February 2020

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Cyclin-dependent kinase-associated proteins Cks1 and Cks2 are essential during early embryogenesis and for cell cycle progression in somatic cells

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Oncogenic Kras requires simultaneous PI3K signaling to induce ERK activation and transform thyroid epithelial cells in vivo

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c-Myc-regulated microRNAs modulate E2F1 expression

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Isolation of a human cyclin cDNA: evidence for cyclin mRNA and protein regulation in the cell cycle and for interaction with p34cdc2

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MiR-21 is an EGFR-regulated anti-apoptotic factor in lung cancer in never-smokers

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An autoregulatory loop mediated by miR-21 and PDCD4 controls the AP-1 activity in RAS transformation

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