Molecular and functional changes in voltage-dependent Na(+) channels following pilocarpine-induced status epilepticus in rat dentate granule cells (Q39650445)

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scientific article published on January 2003

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English	Molecular and functional changes in voltage-dependent Na(+) channels following pilocarpine-induced status epilepticus in rat dentate granule cells	scientific article published on January 2003

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scholarly article

1 reference

Europe PubMed Central

PubMed publication ID

13 September 2017

Molecular and functional changes in voltage-dependent Na(+) channels following pilocarpine-induced status epilepticus in rat dentate granule cells (English)

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Europe PubMed Central

PubMed publication ID

13 September 2017

status epilepticus

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inferred from title

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author name string

Ellerkmann RK

1

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Europe PubMed Central

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13 September 2017

Remy S

2

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Europe PubMed Central

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13 September 2017

Chen J

3

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Europe PubMed Central

PubMed publication ID

13 September 2017

Sochivko D

4

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Europe PubMed Central

PubMed publication ID

13 September 2017

Elger CE

5

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Europe PubMed Central

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13 September 2017

Urban BW

6

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Europe PubMed Central

PubMed publication ID

13 September 2017

Becker A

7

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Europe PubMed Central

PubMed publication ID

13 September 2017

Beck H

8

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Europe PubMed Central

PubMed publication ID

13 September 2017

language of work or name

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publication date

1 January 2003

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Europe PubMed Central

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13 September 2017

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Europe PubMed Central

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13 September 2017

119

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Europe PubMed Central

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13 September 2017

2

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13 September 2017

323-333

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Europe PubMed Central

PubMed publication ID

13 September 2017

Induction of neonatal sodium channel II and III alpha-isoform mRNAs in neurons and microglia after status epilepticus in the rat hippocampus.

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Liquid junction potentials and small cell effects in patch-clamp analysis

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Changes in the mRNAs encoding subtypes I, II and III sodium channel alpha subunits following kainate-induced seizures in rat brain

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Molecular cloning, distribution and functional analysis of the NA(V)1.6. Voltage-gated sodium channel from human brain

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Cellular and molecular biology of voltage-gated sodium channels

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Long-term effects of pilocarpine in rats: structural damage of the brain triggers kindling and spontaneous recurrent seizures.

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Axonal action-potential initiation and Na+ channel densities in the soma and axon initial segment of subicular pyramidal neurons

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Slow recovery from inactivation regulates the availability of voltage-dependent Na(+) channels in hippocampal granule cells, hilar neurons and basket cells

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Increase in mRNAs encoding neonatal II and III sodium channel alpha-isoforms during kainate-induced seizures in adult rat hippocampus

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mRNA coding for voltage-gated sodium channel beta2 subunit in rat central nervous system: cellular distribution and changes following kainate-induced seizures

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Dendritic sodium spikes are variable triggers of axonal action potentials in hippocampal CA1 pyramidal neurons

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Primary structure and functional expression of the beta 1 subunit of the rat brain sodium channel

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Functional co-expression of the beta 1 and type IIA alpha subunits of sodium channels in a mammalian cell line

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Spike after-depolarization and burst generation in adult rat hippocampal CA1 pyramidal cells.

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Prolonged sodium channel inactivation contributes to dendritic action potential attenuation in hippocampal pyramidal neurons

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A gain-of-function mutation in the sodium channel gene Scn2a results in seizures and behavioral abnormalities

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Sodium currents in isolated rat CA1 pyramidal and dentate granule neurones in the post-status epilepticus model of epilepsy

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Altered brain sodium channel transcript levels in human epilepsy

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Phenytoin and carbamazepine: potential- and frequency-dependent block of Na currents in mammalian myelinated nerve fibers

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Action potential initiation and propagation in rat neocortical pyramidal neurons

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Identifiers

10.1016/S0306-4522(03)00168-4

1 reference

Europe PubMed Central

PubMed publication ID

13 September 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

13 September 2017

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