The receptor for advanced glycation end products (RAGE) is a central mediator of the interaction of AGE-beta2microglobulin with human mononuclear phagocytes via an oxidant-sensitive pathway. Implications for the pathogenesis of dialysis-related amyl (Q39770000)

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English	The receptor for advanced glycation end products (RAGE) is a central mediator of the interaction of AGE-beta2microglobulin with human mononuclear phagocytes via an oxidant-sensitive pathway. Implications for the pathogenesis of dialysis-related amyl	scientific article

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scholarly article

1 reference

Europe PubMed Central

PMC publication ID

14 September 2017

The receptor for advanced glycation end products (RAGE) is a central mediator of the interaction of AGE-beta2microglobulin with human mononuclear phagocytes via an oxidant-sensitive pathway. Implications for the pathogenesis of dialysis-related amyl (English)

1 reference

Europe PubMed Central

PMC publication ID

14 September 2017

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author name string

T Miyata

1

1 reference

Europe PubMed Central

PMC publication ID

14 September 2017

O Hori

2

1 reference

Europe PubMed Central

PMC publication ID

14 September 2017

J Zhang

3

1 reference

Europe PubMed Central

PMC publication ID

14 September 2017

S D Yan

4

1 reference

Europe PubMed Central

PMC publication ID

14 September 2017

L Ferran

5

1 reference

Europe PubMed Central

PMC publication ID

14 September 2017

Y Iida

6

1 reference

Europe PubMed Central

PMC publication ID

14 September 2017

A M Schmidt

7

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Europe PubMed Central

PMC publication ID

14 September 2017

language of work or name

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publication date

1 September 1996

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Europe PubMed Central

PMC publication ID

14 September 2017

Journal of Clinical Investigation

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Europe PubMed Central

PMC publication ID

14 September 2017

98

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Europe PubMed Central

PMC publication ID

14 September 2017

5

1 reference

Europe PubMed Central

PMC publication ID

14 September 2017

1088-1094

1 reference

Europe PubMed Central

PMC publication ID

14 September 2017

Receptor-mediated endothelial cell dysfunction in diabetic vasculopathy. Soluble receptor for advanced glycation end products blocks hyperpermeability in diabetic rats

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=507528

22 September 2017

Regulation of human mononuclear phagocyte migration by cell surface-binding proteins for advanced glycation end products

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https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=507528

22 September 2017

beta 2-Microglobulin modified with advanced glycation end products is a major component of hemodialysis-associated amyloidosis

1 reference

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22 September 2017

Lipid advanced glycosylation: pathway for lipid oxidation in vivo

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=507528

22 September 2017

Survey of the distribution of a newly characterized receptor for advanced glycation end products in tissues.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=507528

22 September 2017

Advanced Maillard reaction end products are associated with Alzheimer disease pathology

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=507528

22 September 2017

Enhanced cellular oxidant stress by the interaction of advanced glycation end products with their receptors/binding proteins.

1 reference

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22 September 2017

Involvement of beta 2-microglobulin modified with advanced glycation end products in the pathogenesis of hemodialysis-associated amyloidosis. Induction of human monocyte chemotaxis and macrophage secretion of tumor necrosis factor-alpha and interleu

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https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=507528

22 September 2017

Receptor for advanced glycation end products (AGEs) has a central role in vessel wall interactions and gene activation in response to circulating AGE proteins

1 reference

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22 September 2017

Glycated tau protein in Alzheimer disease: a mechanism for induction of oxidant stress

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22 September 2017

The receptor for advanced glycation end products (RAGE) is a cellular binding site for amphoterin. Mediation of neurite outgrowth and co-expression of rage and amphoterin in the developing nervous system

1 reference

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22 September 2017

Advanced glycation endproducts interacting with their endothelial receptor induce expression of vascular cell adhesion molecule-1 (VCAM-1) in cultured human endothelial cells and in mice. A potential mechanism for the accelerated vasculopathy of dia

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=507528

22 September 2017

Advanced glycosylation end products in tissue and the biochemical basis of diabetic complications

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=507528

22 September 2017

Toward a Maillard reaction theory of aging.

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https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=507528

22 September 2017

Structure elucidation of a senescence cross-link from human extracellular matrix. Implication of pentoses in the aging process

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=507528

22 September 2017

Role of oxidative stress in development of complications in diabetes

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=507528

22 September 2017

Beta-2-microglobulin amyloidosis and renal bone disease

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=507528

22 September 2017

Two novel rat liver membrane proteins that bind advanced glycosylation endproducts: relationship to macrophage receptor for glucose-modified proteins

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=507528

22 September 2017

Glucose and diabetic vascular disease.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=507528

22 September 2017

Integrins: versatility, modulation, and signaling in cell adhesion

1 reference

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22 September 2017

17 beta-estradiol inhibits interleukin-6 production by bone marrow-derived stromal cells and osteoblasts in vitro: a potential mechanism for the antiosteoporotic effect of estrogens

1 reference

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22 September 2017

Cloning and expression of a cell surface receptor for advanced glycosylation end products of proteins

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=507528

22 September 2017

Collagenase Immunolocalization in Cultures of Rheumatoid Synovial Cells

1 reference

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22 September 2017

Cytokines and estrogen in bone: anti-osteoporotic effects

1 reference

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25 September 2017

Autoxidative glycosylation and possible involvement of peroxides and free radicals in LDL modification by glucose

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https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=507528

25 September 2017

Changes in cell shape correlate with collagenase gene expression in rabbit synovial fibroblasts

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25 September 2017

Expression of receptors for advanced glycation end products in peripheral occlusive vascular disease

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=507528

20 August 2018

Immunological evidence for the presence of advanced glycosylation end products in atherosclerotic lesions of euglycemic rabbits

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https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=507528

20 August 2018

Non-enzymatically glycated tau in Alzheimer's disease induces neuronal oxidant stress resulting in cytokine gene expression and release of amyloid beta-peptide.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=507528

20 August 2018

Protein iodination with solid state lactoperoxidase

1 reference

https://pubmed.ncbi.nlm.nih.gov/8787669

12 December 2020

based on heuristic

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Catalysis of lipid peroxidation by glucose and glycosylated collagen

1 reference

https://pubmed.ncbi.nlm.nih.gov/8787669

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Dialysis-related amyloidosis

1 reference

https://pubmed.ncbi.nlm.nih.gov/8787669

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Isolation and characterization of two binding proteins for advanced glycosylation end products from bovine lung which are present on the endothelial cell surface

1 reference

https://pubmed.ncbi.nlm.nih.gov/8787669

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Parathyroid hormone (PTH) and PTH-like protein (PLP) stimulate interleukin-6 production by osteogenic cells: a possible role of interleukin-6 in osteoclastogenesis

1 reference

https://pubmed.ncbi.nlm.nih.gov/8787669

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based on heuristic

inferred from PubMed ID database lookup

Macrophages adhere to glucose-modified basement membrane collagen IV via their scavenger receptors

1 reference

https://pubmed.ncbi.nlm.nih.gov/8787669

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

O2- generation and lipid peroxidation during the oxidation of a glycated polypeptide, glycated polylysine, in the presence of iron-ADP

1 reference

https://pubmed.ncbi.nlm.nih.gov/8787669

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Immunochemical approach to characterize advanced glycation end products of the Maillard reaction. Evidence for the presence of a common structure

1 reference

https://pubmed.ncbi.nlm.nih.gov/8787669

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Identifiers

10.1172/JCI118889

1 reference

Europe PubMed Central

PMC publication ID

14 September 2017

PMC publication ID

1 reference

Europe PubMed Central

PMC publication ID

14 September 2017

PubMed publication ID

1 reference

Europe PubMed Central

PMC publication ID

14 September 2017

ResearchGate publication ID

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