Chronic nicotine exposure reduces N-methyl-D-aspartate receptor-mediated damage in the hippocampus without altering calcium accumulation or extrusion: evidence of calbindin-D28K overexpression (Q32065508)

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English	Chronic nicotine exposure reduces N-methyl-D-aspartate receptor-mediated damage in the hippocampus without altering calcium accumulation or extrusion: evidence of calbindin-D28K overexpression	scientific article

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scholarly article

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Europe PubMed Central

PubMed publication ID

6 July 2017

Chronic nicotine exposure reduces N-methyl-D-aspartate receptor-mediated damage in the hippocampus without altering calcium accumulation or extrusion: evidence of calbindin-D28K overexpression (English)

1 reference

Europe PubMed Central

PubMed publication ID

6 July 2017

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Prendergast MA

1

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Europe PubMed Central

PubMed publication ID

6 July 2017

Harris BR

2

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Europe PubMed Central

PubMed publication ID

6 July 2017

Mayer S

3

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Europe PubMed Central

PubMed publication ID

6 July 2017

Holley RC

4

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Europe PubMed Central

PubMed publication ID

6 July 2017

Hauser KF

5

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Europe PubMed Central

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6 July 2017

Littleton JM

6

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Europe PubMed Central

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6 July 2017

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1 January 2001

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Europe PubMed Central

PubMed publication ID

6 July 2017

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Europe PubMed Central

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6 July 2017

102

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Europe PubMed Central

PubMed publication ID

6 July 2017

1

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Europe PubMed Central

PubMed publication ID

6 July 2017

75-85

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Europe PubMed Central

PubMed publication ID

6 July 2017

Mechanisms and effects of intracellular calcium buffering on neuronal survival in organotypic hippocampal cultures exposed to anoxia/aglycemia or to excitotoxins.

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Effects of nicotinic agonists on the NMDA receptor

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Nicotine-induced protection of cultured cortical neurons against N-methyl-D-aspartate receptor-mediated glutamate cytotoxicity

1 reference

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Nicotinic acetylcholine receptors on hippocampal neurons: distribution on the neuronal surface and modulation of receptor activity

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The ability of amyloid beta-protein [A beta P (1-40)] to form Ca2+ channels provides a mechanism for neuronal death in Alzheimer's disease.

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Sources of variability in nicotine and cotinine levels with use of nicotine nasal spray, transdermal nicotine, and cigarette smoking

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Calcium transport and buffering in neurons

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Calbindin-D 28K in hippocampal organotypic cultures

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Comparison of the regional expression of nicotinic acetylcholine receptor alpha7 mRNA and [125I]-alpha-bungarotoxin binding in human postmortem brain

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Modulation of synaptic transmission by nicotine and nicotinic antagonists in hippocampus.

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Ionic dependence of glutamate neurotoxicity

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Excitotoxic cell death

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Rat calbindin D28K: purification, quantitation, immunocytochemical localization, and comparative aspects

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Altered behavioral sensitivity of Ca(2+)-modulating drugs after chronic nicotine administration in mice

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The quantity of calcium that appears to induce neuronal death.

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Concentration- and cell type-specific effects of calbindin D28k on vulnerability of hippocampal neurons to seizure-induced injury

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Possible Causes of Alzheimer's Disease: Amyloid Fragments, Free Radicals, and Calcium Homeostasis

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The presence of calbindin in rat cortical neurons protects in vitro from oxydative stress

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Ionized Intracellular Calcium Concentration Predicts Excitotoxic Neuronal Death: Observations with Low-Affinity Fluorescent Calcium Indicators

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Nicotine protects cultured cortical neurons against glutamate-induced cytotoxicity via alpha7-neuronal receptors and neuronal CNS receptors

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Stimulation of alpha4beta2 nicotinic acetylcholine receptors inhibits beta-amyloid toxicity.

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Nicotinic receptor stimulation protects neurons against beta-amyloid toxicity.

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NMDA-dependent superoxide production and neurotoxicity

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Calcium homeostasis in ageing: Studies on the calcium binding protein calbindin D28K

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Inactivation of NMDA channels in cultured hippocampal neurons by intracellular calcium

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Stimulation of a nicotinic ACh receptor causes depolarization and activation of L-type Ca2+ channels in rat pinealocytes.

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Current concepts of ethanol dependence

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Identifiers

10.1016/S0306-4522(00)00450-4

1 reference

Europe PubMed Central

PubMed publication ID

6 July 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

6 July 2017

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