Biphasic effects of copper on neurotransmission in rat hippocampal neurons. (Q46176771)

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9 February 2020

title

Biphasic effects of copper on neurotransmission in rat hippocampal neurons (English)

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9 February 2020

2

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Giancarlo V De Ferrari

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9 February 2020

Carlos Opazo

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author name string

Christian Peters

1

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9 February 2020

Fernando J Sepúlveda

3

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9 February 2020

Juan Urrutia

4

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9 February 2020

Mauricio Quiroz

5

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Sandra Luza

6

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9 February 2020

Luis G Aguayo

8

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language of work or name

English

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publication date

1 September 2011

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Journal of Neurochemistry

9 February 2020

published in

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volume

119

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9 February 2020

issue

1

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page(s)

78-88

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Canonical Wnt3a modulates intracellular calcium and enhances excitatory neurotransmission in hippocampal neurons

9 February 2020

cites work

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Nerve endings from rat brain tissue release copper upon depolarization. A possible role in regulating neuronal excitability

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Copper suppresses hippocampus LTP in the rat, but does not alter learning or memory in the morris water maze.

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Copper, iron and zinc in Alzheimer's disease senile plaques

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Zinc and copper: pharmacological probes and endogenous modulators of neuronal excitability.

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Surface expression of AMPA receptors in hippocampal neurons is regulated by an NSF-dependent mechanism.

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Ctr1 is an apical copper transporter in mammalian intestinal epithelial cells in vivo that is controlled at the level of protein stability

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NMDA receptor activation mediates copper homeostasis in hippocampal neurons

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Role of the Menkes copper-transporting ATPase in NMDA receptor-mediated neuronal toxicity

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Direct interactions between PSD-95 and stargazin control synaptic AMPA receptor number

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High-affinity copper block of GABA(A) receptor-mediated currents in acutely isolated cerebellar Purkinje cells of the rat.

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The effects of copper ions on glutamate receptors in cultured rat cortical neurons

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