Complement activation following reoxygenation of hypoxic human endothelial cells: role of intracellular reactive oxygen species, NF-kappaB and new protein synthesis (Q47832213)

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scientific article published in March 1998

Language	Label	Description	Also known as
English	Complement activation following reoxygenation of hypoxic human endothelial cells: role of intracellular reactive oxygen species, NF-kappaB and new protein synthesis	scientific article published in March 1998

Statements

scholarly article

1 reference

Europe PubMed Central

PubMed publication ID

6 February 2018

http://europepmc.org/abstract/MED/9667422

Complement activation following reoxygenation of hypoxic human endothelial cells: role of intracellular reactive oxygen species, NF-kappaB and new protein synthesis (English)

1 reference

Europe PubMed Central

PubMed publication ID

6 February 2018

http://europepmc.org/abstract/MED/9667422

0 references

reactive oxygen species

1 reference

based on heuristic

inferred from title

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based on heuristic

inferred from title

author name string

C D Collard

1

1 reference

Europe PubMed Central

PubMed publication ID

6 February 2018

http://europepmc.org/abstract/MED/9667422

A Agah

2

1 reference

Europe PubMed Central

PubMed publication ID

6 February 2018

http://europepmc.org/abstract/MED/9667422

G L Stahl

3

1 reference

Europe PubMed Central

PubMed publication ID

6 February 2018

http://europepmc.org/abstract/MED/9667422

publication date

1 March 1998

1 reference

Europe PubMed Central

PubMed publication ID

6 February 2018

http://europepmc.org/abstract/MED/9667422

number of pages

12

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inferred from page(s)

Immunopharmacology

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Europe PubMed Central

PubMed publication ID

6 February 2018

http://europepmc.org/abstract/MED/9667422

39

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Europe PubMed Central

PubMed publication ID

6 February 2018

http://europepmc.org/abstract/MED/9667422

1

1 reference

Europe PubMed Central

PubMed publication ID

6 February 2018

http://europepmc.org/abstract/MED/9667422

39-50

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Europe PubMed Central

PubMed publication ID

6 February 2018

http://europepmc.org/abstract/MED/9667422

Stimulation-dependent I kappa B alpha phosphorylation marks the NF-kappa B inhibitor for degradation via the ubiquitin-proteasome pathway

1 reference

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Alternative pathway activation of complement by cultured human proximal tubular epithelial cells.

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Cardioprotective effects of a C1 esterase inhibitor in myocardial ischemia and reperfusion

1 reference

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Reoxygenation of Hypoxic Human Umbilical Vein Endothelial Cells Activates the Classic Complement Pathway

1 reference

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Can superoxide dismutase alter myocardial infarct size?

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Dihydrorhodamine 123: a fluorescent probe for superoxide generation?

1 reference

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Hypoxia/reoxygenation increases the permeability of endothelial cell monolayers: role of oxygen radicals

1 reference

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The complement system in myocardial ischaemia/reperfusion injury

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Sulfhydryl compounds, captopril, and MPG inhibit complement-mediated myocardial injury

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Complement-induced endothelial dysfunction in rabbits: mechanisms, recovery, and gender differences

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Inhibition of nuclear translocation of transcription factor NF-kappa B by a synthetic peptide containing a cell membrane-permeable motif and nuclear localization sequence

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Human umbilical vein endothelial cells submitted to hypoxia-reoxygenation in vitro: implication of free radicals, xanthine oxidase, and energy deficiency.

1 reference

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Hypoxia induces endothelial cell synthesis of membrane-associated proteins

1 reference

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The ubiquitin-proteasome pathway is required for processing the NF-kappa B1 precursor protein and the activation of NF-kappa B

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Evaluation of 2',7'-dichlorofluorescin and dihydrorhodamine 123 as fluorescent probes for intracellular H2O2 in cultured endothelial cells

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Hypoxia induces cyclooxygenase-2 via the NF-kappaB p65 transcription factor in human vascular endothelial cells

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Reactive oxygen intermediates as apparently widely used messengers in the activation of the NF-kappa B transcription factor and HIV-1

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Hydrogen peroxide-induced cardiovascular reflexes. Role of hydroxyl radicals

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Activation of the fifth component of human complement by oxygen-derived free radicals, and by methionine oxidizing agents: a comparison

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Oxidants generated by the myeloperoxidase-halide system activate the fifth component of human complement, C5

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Reperfusion injury of ischemic skeletal muscle is mediated by natural antibody and complement

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Soluble human complement receptor type 1: in vivo inhibitor of complement suppressing post-ischemic myocardial inflammation and necrosis

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A Striking Similarity in the Organization of the E-Selectin and Bate Interferon gene Promoters

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Hypoxia enhances induction of endothelial ICAM-1: role for metabolic acidosis and proteasomes.

1 reference

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Identifiers

10.1016/S0162-3109(97)00096-9

1 reference

Europe PubMed Central

PubMed publication ID

6 February 2018

http://europepmc.org/abstract/MED/9667422

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

6 February 2018

http://europepmc.org/abstract/MED/9667422

ResearchGate publication ID

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