Expanded polyglutamine protein forms nuclear inclusions and causes neural degeneration in Drosophila (Q47945602)

7 February 2018

title

Expanded polyglutamine protein forms nuclear inclusions and causes neural degeneration in Drosophila (English)

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7 February 2018

author

Nancy M. Bonini

7

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7 February 2018

author name string

J M Warrick

1

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7 February 2018

H L Paulson

2

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7 February 2018

G L Gray-Board

3

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7 February 2018

Q T Bui

4

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7 February 2018

K H Fischbeck

5

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7 February 2018

R N Pittman

6

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7 February 2018

language of work or name

English

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publication date

1 June 1998

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7 February 2018

published in

Cell

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7 February 2018

volume

93

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7 February 2018

issue

6

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7 February 2018

page(s)

939-949

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7 February 2018

Rethinking genotype and phenotype correlations in polyglutamine expansion disorders

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Formation of neuronal intranuclear inclusions underlies the neurological dysfunction in mice transgenic for the HD mutation

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Aggregation of huntingtin in neuronal intranuclear inclusions and dystrophic neurites in brain

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Spinocerebellar ataxia 3 and Machado-Joseph disease: clinical, molecular, and neuropathological features

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Reiterative use of the EGF receptor triggers differentiation of all cell types in the Drosophila eye.

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The head involution defective gene of Drosophila melanogaster functions in programmed cell death

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Suppression of aggregate formation and apoptosis by transglutaminase inhibitors in cells expressing truncated DRPLA protein with an expanded polyglutamine stretch.

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Expanded polyglutamine in the Machado-Joseph disease protein induces cell death in vitro and in vivo

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CAG expansions in a novel gene for Machado-Joseph disease at chromosome 14q32.1

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Ectopic and increased expression of Fasciclin II alters motoneuron growth cone guidance

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Exon 1 of the HD gene with an expanded CAG repeat is sufficient to cause a progressive neurological phenotype in transgenic mice

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The cerebellar leucine-rich acidic nuclear protein interacts with ataxin-1

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The activities of two Ets-related transcription factors required for Drosophila eye development are modulated by the Ras/MAPK pathway

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Ectopically expressed CAG repeats cause intranuclear inclusions and a progressive late onset neurological phenotype in the mouse

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7 January 2021

Machado-Joseph disease gene product is a cytoplasmic protein widely expressed in brain

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Intranuclear inclusions of expanded polyglutamine protein in spinocerebellar ataxia type 3.

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The complex pathology of trinucleotide repeats

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The locus elav of Drosophila melanogaster is expressed in neurons at all developmental stages.

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Intranuclear neuronal inclusions: a common pathogenic mechanism for glutamine-repeat neurodegenerative diseases?

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Huntingtin-encoded polyglutamine expansions form amyloid-like protein aggregates in vitro and in vivo

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Trinucleotide expansion within the MJD1 gene presents clinically as spinocerebellar ataxia and occurs most frequently in German SCA patients

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7 January 2021

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Identifiers

DOI

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7 February 2018

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7 February 2018