Amyotrophic lateral sclerosis: current issues in classification, pathogenesis and molecular pathology (Q47950253)

7 February 2018

review article

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title

Amyotrophic lateral sclerosis: current issues in classification, pathogenesis and molecular pathology (English)

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7 February 2018

amyotrophic lateral sclerosis

main subject

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3

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author name string

Ince PG

series ordinal

1

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Lowe J

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2

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publication date

1 April 1998

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Neuropathology and Applied Neurobiology

published in

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volume

24

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issue

2

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7 February 2018

page(s)

104-117

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7 February 2018

Superoxide dismutase in familial amyotrophic lateral sclerosis: models for gain of function

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Colocalization of NOS and SOD1 in neurofilament accumulation within motor neurons of amyotrophic lateral sclerosis: an immunohistochemical study

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Mutations in copper-zinc superoxide dismutase that cause amyotrophic lateral sclerosis alter the zinc binding site and the redox behavior of the protein

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Apolipoprotein E genotyping in sporadic amyotrophic lateral sclerosis: evidence for a major influence on the clinical presentation and prognosis

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Molecular genetic basis of familial ALS.

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Transgenic mice carrying a human mutant superoxide dismutase transgene develop neuronal cytoskeletal pathology resembling human amyotrophic lateral sclerosis lesions

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Hippocampal and neocortical ubiquitin-immunoreactive inclusions in amyotrophic lateral sclerosis with dementia

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An adverse property of a familial ALS-linked SOD1 mutation causes motor neuron disease characterized by vacuolar degeneration of mitochondria

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A familial amyotrophic lateral sclerosis-associated A4V Cu, Zn-superoxide dismutase mutant has a lower Km for hydrogen peroxide. Correlation between clinical severity and the Km value.

21 January 2018

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10.1046/J.1365-2990.1998.00108.X

21 January 2018

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DOI

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7 February 2018

1 reference

7 February 2018