Inhibition of interneuron firing extends the spread of endocannabinoid signaling in the cerebellum (Q48581988)

13 February 2018

title

Inhibition of interneuron firing extends the spread of endocannabinoid signaling in the cerebellum (English)

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13 February 2018

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Anatol C Kreitzer

1

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13 February 2018

Adam G Carter

series ordinal

2

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13 February 2018

Wade G Regehr

series ordinal

3

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13 February 2018

language of work or name

English

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publication date

1 May 2002

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13 February 2018

published in

Neuron

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13 February 2018

volume

34

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13 February 2018

issue

5

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13 February 2018

page(s)

787-796

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13 February 2018

Supersensitivity to anandamide and enhanced endogenous cannabinoid signaling in mice lacking fatty acid amide hydrolase

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Short-term retrograde inhibition of GABAergic synaptic currents in rat Purkinje cells is mediated by endogenous cannabinoids.

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A new perspective on cannabinoid signalling: complementary localization of fatty acid amide hydrolase and the CB1 receptor in rat brain

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Cerebellar depolarization-induced suppression of inhibition is mediated by endogenous cannabinoids.

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Retrograde inhibition of presynaptic calcium influx by endogenous cannabinoids at excitatory synapses onto Purkinje cells

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Unresponsiveness to cannabinoids and reduced addictive effects of opiates in CB1 receptor knockout mice

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Cannabinoids activate an inwardly rectifying potassium conductance and inhibit Q-type calcium currents in AtT20 cells transfected with rat brain cannabinoid receptor.

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Fatty acid amide hydrolase is located preferentially in large neurons in the rat central nervous system as revealed by immunohistochemistry

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Neighboring cerebellar Purkinje cells communicate via retrograde inhibition of common presynaptic interneurons

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Endogenous cannabinoids mediate retrograde signalling at hippocampal synapses

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Presynaptic specificity of endocannabinoid signaling in the hippocampus

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Increased mortality, hypoactivity, and hypoalgesia in cannabinoid CB1 receptor knockout mice

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7 January 2021

10.1016/S0896-6273(02)00695-5

Identifiers

DOI

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13 February 2018

1 reference

13 February 2018