Evasion of early cellular response mechanisms following low level radiation-induced DNA damage. (Q40514065)

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1 January 2020

title

Evasion of early cellular response mechanisms following low level radiation-induced DNA damage (English)

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1

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1 January 2020

author name string

Julie M Schwaninger

2

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1 January 2020

Alfred J Ntambi

3

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1 January 2020

Thomas W Keller

4

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1 January 2020

William G Nelson

5

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1 January 2020

Larry E Dillehay

6

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1 January 2020

Theodore L Deweese

7

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language of work or name

English

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publication date

17 September 2004

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Journal of Biological Chemistry

1 January 2020

published in

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volume

279

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issue

48

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page(s)

49624-49632

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Genome maintenance mechanisms for preventing cancer

1 January 2020

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Chromosomal stability and the DNA double-stranded break connection

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Sensing and repairing DNA double-strand breaks

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Endogenous DNA double-strand breaks: Production, fidelity of repair, and induction of cancer

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Inverse radiation dose-rate effects on somatic and germ-line mutations and DNA damage rates

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Low-dose hypersensitivity: current status and possible mechanisms.

21 January 2018

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DNA damage activates ATM through intermolecular autophosphorylation and dimer dissociation

21 January 2018

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Requirement of the MRN complex for ATM activation by DNA damage

21 January 2018

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Distinct functional domains of Nbs1 modulate the timing and magnitude of ATM activation after low doses of ionizing radiation

21 January 2018

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Direct activation of the ATM protein kinase by the Mre11/Rad50/Nbs1 complex

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ATM phosphorylates histone H2AX in response to DNA double-strand breaks

21 January 2018

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A critical role for histone H2AX in recruitment of repair factors to nuclear foci after DNA damage

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Genomic instability in mice lacking histone H2AX

21 January 2018

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Increased ionizing radiation sensitivity and genomic instability in the absence of histone H2AX

21 January 2018

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Expression of phosphorylated histone H2AX in cultured cell lines following exposure to X-rays.

21 January 2018

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Evidence for a lack of DNA double-strand break repair in human cells exposed to very low x-ray doses

21 January 2018

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Effects of exposure to low-dose-rate (60)co gamma rays on human tumor cells in vitro.

21 January 2018

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Phosphorylation of histone H2AX as a measure of radiosensitivity

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DNA double-stranded breaks induce histone H2AX phosphorylation on serine 139

21 January 2018

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Megabase chromatin domains involved in DNA double-strand breaks in vivo

21 January 2018

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DNA damage-induced G2-M checkpoint activation by histone H2AX and 53BP1

21 January 2018

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DNA-PK is activated by nucleosomes and phosphorylates H2AX within the nucleosomes in an acetylation-dependent manner

21 January 2018

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ATM and DNA-PK function redundantly to phosphorylate H2AX after exposure to ionizing radiation

21 January 2018

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Two molecularly distinct G(2)/M checkpoints are induced by ionizing irradiation

21 January 2018

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An association between the radiation-induced arrest of G2-phase cells and low-dose hyper-radiosensitivity: a plausible underlying mechanism?

21 January 2018

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Low-dose hyper-radiosensitivity: a consequence of ineffective cell cycle arrest of radiation-damaged G2-phase cells.

21 January 2018

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Protective packaging for DNA

21 January 2018

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