High levels of Cre expression in neuronal progenitors cause defects in brain development leading to microencephaly and hydrocephaly. (Q41919230)

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30 December 2019

title

High levels of Cre expression in neuronal progenitors cause defects in brain development leading to microencephaly and hydrocephaly (English)

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Paolo Emanuele Forni

1

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Riccardo Taulli

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Alessandro Vercelli

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author name string

Claudio Scuoppo

2

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30 December 2019

Itaru Imayoshi

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Walter Dastrù

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30 December 2019

Ulrich A K Betz

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Patrizia Muzzi

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Daniela Martinuzzi

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Ryoichiro Kageyama

11

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Carola Ponzetto

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language of work or name

English

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publication date

1 September 2006

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published in

Journal of Neuroscience

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volume

26

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issue

37

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30 December 2019

page(s)

9593-9602

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describes a project that uses

30 December 2019

ImageJ

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11 June 2022

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inferred from PubMed Central ID database lookup

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Disruption of the glucocorticoid receptor gene in the nervous system results in reduced anxiety

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A programmed ependymal denudation precedes congenital hydrocephalus in the hyh mutant mouse

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Efficient recombination in diverse tissues by a tamoxifen-inducible form of Cre: a tool for temporally regulated gene activation/inactivation in the mouse

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Congenital hydrocephalus in hy3 mice is caused by a frameshift mutation in Hydin, a large novel gene

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Chromosome segregation defects contribute to aneuploidy in normal neural progenitor cells.

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Impaired brain angiogenesis and neuronal apoptosis induced by conditional homozygous inactivation of vascular endothelial growth factor

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The neuregulin receptor, ErbB4, is not required for normal development and adult maintenance of the substantia nigra pars compacta.

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The chromatin-remodeling protein ATRX is critical for neuronal survival during corticogenesis

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Ependymal denudation and alterations of the subventricular zone occur in human fetuses with a moderate communicating hydrocephalus

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Alcohol inhibits cell-cell adhesion mediated by human L1

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Ligand-activated site-specific recombination in mice

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Bypass of lethality with mosaic mice generated by Cre– loxP -mediated recombination

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L1 knockout mice show dilated ventricles, vermis hypoplasia and impaired exploration patterns.

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Generalized lacZ expression with the ROSA26 Cre reporter strain

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10.1523/JNEUROSCI.2815-06.2006

Identifiers

DOI

1 reference

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30 December 2019

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30 December 2019

PubMed publication ID

16971543

1 reference