Extracellular redox status regulates Nrf2 activation through mitochondrial reactive oxygen species (Q42833679)

8 November 2017

title

Extracellular redox status regulates Nrf2 activation through mitochondrial reactive oxygen species (English)

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8 November 2017

main subject

reactive oxygen species

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Barry R Imhoff

1

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8 November 2017

Jason M Hansen

series ordinal

2

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8 November 2017

language of work or name

English

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publication date

10 December 2009

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8 November 2017

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8 November 2017

volume

424

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8 November 2017

issue

3

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8 November 2017

page(s)

491-500

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Nuclear and mitochondrial compartmentation of oxidative stress and redox signaling

8 November 2017

cites work

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Extracellular redox state: refining the definition of oxidative stress in aging

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Nuclear and cytoplasmic peroxiredoxin-1 differentially regulate NF-kappaB activities

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Mitochondrial thioredoxin-2 has a key role in determining tumor necrosis factor-alpha-induced reactive oxygen species generation, NF-kappaB activation, and apoptosis

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Redox imbalance in cystine/glutamate transporter-deficient mice

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Extracellular thiol/disulfide redox state affects proliferation rate in a human colon carcinoma (Caco2) cell line

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Intracellular proatherogenic events and cell adhesion modulated by extracellular thiol/disulfide redox state

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NRF2 and KEAP1 mutations: permanent activation of an adaptive response in cancer

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Keap1-dependent proteasomal degradation of transcription factor Nrf2 contributes to the negative regulation of antioxidant response element-driven gene expression

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Nrf2, a Cap'n'Collar transcription factor, regulates induction of the heme oxygenase-1 gene

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Human prx1 gene is a target of Nrf2 and is up-regulated by hypoxia/reoxygenation: implication to tumor biology

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Nrf1 and Nrf2 positively and c-Fos and Fra1 negatively regulate the human antioxidant response element-mediated expression of NAD(P)H:quinone oxidoreductase1 gene

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Electrophile response element-mediated induction of the cystine/glutamate exchange transporter gene expression

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Nrf2 transcriptionally activates the mafG gene through an antioxidant response element

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Antioxidant supplements prevent oxidation of cysteine/cystine redox in patients with age-related macular degeneration

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Hydrogen peroxide mediates the cell growth and transformation caused by the mitogenic oxidase Nox1

21 January 2018

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The absence of mitochondrial thioredoxin 2 causes massive apoptosis, exencephaly, and early embryonic lethality in homozygous mice

21 January 2018

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Transcription factor Nrf2 mediates an adaptive response to sulforaphane that protects fibroblasts in vitro against the cytotoxic effects of electrophiles, peroxides and redox-cycling agents

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Role of membrane transport in metabolism and function of glutathione in mammals

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21 January 2018

Identifiers

DOI

10.1042/BJ20091286

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8 November 2017

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