The Bloom's syndrome helicase can promote the regression of a model replication fork. (Q54463698)

30 May 2018

title

The Bloom's syndrome helicase can promote the regression of a model replication fork. (English)

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30 May 2018

author name string

Christine Ralf

series ordinal

1

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30 May 2018

Ian D Hickson

series ordinal

2

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30 May 2018

Leonard Wu

series ordinal

3

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30 May 2018

language of work or name

English

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publication date

8 June 2006

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30 May 2018

Journal of Biological Chemistry

published in

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30 May 2018

volume

281

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30 May 2018

issue

32

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30 May 2018

page(s)

22839-22846

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30 May 2018

Suffering in silence: the tolerance of DNA damage

cites work

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M604268200

Fate of DNA replication fork encountering a single DNA lesion during oriC plasmid DNA replication in vitro

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M604268200

Functional uncoupling of twin polymerases: mechanism of polymerase dissociation from a lagging-strand block.

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M604268200

Uncoupling of leading- and lagging-strand DNA replication during lesion bypass in vivo

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M604268200

Differential replication of a single, UV-induced lesion in the leading or lagging strand by a human cell extract: fork uncoupling or gap formation

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M604268200

Recombinational DNA repair of damaged replication forks in Escherichia coli: questions

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M604268200

Genome stability and the processing of damaged replication forks by RecG.

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M604268200

RecA protein promotes the regression of stalled replication forks in vitro

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M604268200

Situational repair of replication forks: roles of RecG and RecA proteins

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M604268200

Fork reversal and ssDNA accumulation at stalled replication forks owing to checkpoint defects

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M604268200

Holliday junctions accumulate in replication mutants via a RecA homolog-independent mechanism

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M604268200

RecQ helicases: caretakers of the genome

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M604268200

RecQ helicases: suppressors of tumorigenesis and premature aging.

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M604268200

Conferring substrate specificity to DNA helicases: role of the RecQ HRDC domain.

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M604268200

High-resolution structure of the E.coli RecQ helicase catalytic core

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M604268200

Werner syndrome protein interacts with human flap endonuclease 1 and stimulates its cleavage activity

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M604268200

The three-dimensional structure of the HRDC domain and implications for the Werner and Bloom syndrome proteins

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M604268200

A putative nucleic acid-binding domain in Bloom's and Werner's syndrome helicases

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M604268200

Colocalization, physical, and functional interaction between Werner and Bloom syndrome proteins

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M604268200

The HRDC domain of BLM is required for the dissolution of double Holliday junctions

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M604268200

A manyfold increase in sister chromatid exchanges in Bloom's syndrome lymphocytes

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M604268200

The Bloom's syndrome helicase promotes the annealing of complementary single-stranded DNA

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M604268200

The Bloom's syndrome gene product is a 3'-5' DNA helicase

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M604268200

The Bloom's syndrome gene product promotes branch migration of holliday junctions

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M604268200

RecQ family members combine strand pairing and unwinding activities to catalyze strand exchange

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M604268200

The Bloom's and Werner's syndrome proteins are DNA structure-specific helicases

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M604268200

Substrate specificity of RusA resolvase reveals the DNA structures targeted by RuvAB and RecG in vivo.

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M604268200

Positive torsional strain causes the formation of a four-way junction at replication forks

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M604268200

Role of the Escherichia coli RecQ DNA helicase in SOS signaling and genome stabilization at stalled replication forks.

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M604268200

Sister chromatid exchanges are mediated by homologous recombination in vertebrate cells

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M604268200

The Bloom's syndrome helicase suppresses crossing over during homologous recombination

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M604268200

The Bloom's syndrome gene product interacts with topoisomerase III

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M604268200

Evidence for BLM and Topoisomerase IIIalpha interaction in genomic stability

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M604268200

Phosphorylation of BLM, dissociation from topoisomerase IIIalpha, and colocalization with gamma-H2AX after topoisomerase I-induced replication damage

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M604268200

Cancer predisposition caused by elevated mitotic recombination in Bloom mice

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M604268200

Recql5 and Blm RecQ DNA helicases have nonredundant roles in suppressing crossovers

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M604268200

Functional relation among RecQ family helicases RecQL1, RecQL5, and BLM in cell growth and sister chromatid exchange formation

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M604268200

Multiple Mechanisms Control Chromosome Integrity after Replication Fork Uncoupling and Restart at Irreparable UV Lesions

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M604268200

21 January 2018

Identifiers

DOI

10.1074/JBC.M604268200

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30 May 2018

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30 May 2018