Peripherin is not a contributing factor to motor neuron disease in a mouse model of amyotrophic lateral sclerosis caused by mutant superoxide dismutase. (Q54766411)

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English	Peripherin is not a contributing factor to motor neuron disease in a mouse model of amyotrophic lateral sclerosis caused by mutant superoxide dismutase.	scientific article

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scholarly article

1 reference

Europe PubMed Central

PubMed publication ID

3 June 2018

http://europepmc.org/abstract/MED/12828939

Peripherin is not a contributing factor to motor neuron disease in a mouse model of amyotrophic lateral sclerosis caused by mutant superoxide dismutase. (English)

1 reference

Europe PubMed Central

PubMed publication ID

3 June 2018

http://europepmc.org/abstract/MED/12828939

amyotrophic lateral sclerosis

0 references

Jean-Martin Beaulieu

2

1 reference

Europe PubMed Central

PubMed publication ID

3 June 2018

http://europepmc.org/abstract/MED/12828939

Roxanne Larivière

1

object named as

Roxanne C Larivière

0 references

Jean-Pierre Julien

4

object named as

Jean-Pierre Julien

1 reference

Europe PubMed Central

PubMed publication ID

3 June 2018

http://europepmc.org/abstract/MED/12828939

author name string

Minh Dang Nguyen

3

1 reference

Europe PubMed Central

PubMed publication ID

3 June 2018

http://europepmc.org/abstract/MED/12828939

publication date

1 July 2003

1 reference

Europe PubMed Central

PubMed publication ID

3 June 2018

http://europepmc.org/abstract/MED/12828939

Neurobiology of Disease

1 reference

Europe PubMed Central

PubMed publication ID

3 June 2018

http://europepmc.org/abstract/MED/12828939

13

1 reference

Europe PubMed Central

PubMed publication ID

3 June 2018

http://europepmc.org/abstract/MED/12828939

2

1 reference

Europe PubMed Central

PubMed publication ID

3 June 2018

http://europepmc.org/abstract/MED/12828939

158-166

1 reference

Europe PubMed Central

PubMed publication ID

3 June 2018

http://europepmc.org/abstract/MED/12828939

Late onset of motor neurons in mice overexpressing wild-type peripherin

1 reference

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Induction of peripherin expression in subsets of brain neurons after lesion injury or cerebral ischemia

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7 January 2021

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Selective distribution of the 57 kDa neural intermediate filament protein in the rat CNS.

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ALS-linked SOD1 mutant G85R mediates damage to astrocytes and promotes rapidly progressive disease with SOD1-containing inclusions.

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7 January 2021

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Aggregation and Motor Neuron Toxicity of an ALS-Linked SOD1 Mutant Independent from Wild-Type SOD1

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7 January 2021

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Peripherin and neurofilament protein coexist in spinal spheroids of motor neuron disease

1 reference

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Protective effect of neurofilament heavy gene overexpression in motor neuron disease induced by mutant superoxide dismutase

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Peripherin expression in hippocampal neurons induced by muscle soluble factor(s).

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Differential expression of two neuronal intermediate-filament proteins, peripherin and the low-molecular-mass neurofilament protein (NF-L), during the development of the rat

1 reference

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The expression of the neuronal intermediate filament protein peripherin in the rat embryo

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7 January 2021

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Motor neuron degeneration in mice that express a human Cu,Zn superoxide dismutase mutation

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Cytoskeletal abnormalities in amyotrophic lateral sclerosis: beneficial or detrimental effects?

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7 January 2021

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Functional circuitry in the brain of immune-challenged rats: partial involvement of prostaglandins

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An essential role of interleukin-1beta in mediating NF-kappaB activity and COX-2 transcription in cells of the blood-brain barrier in response to a systemic and localized inflammation but not during endotoxemia

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Reduced number of unmyelinated sensory axons in peripherin null mice

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The mosaic of brain glial hyperactivity during normal ageing and its attenuation by food restriction

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Time course of neuropathology in the spinal cord of G86R superoxide dismutase transgenic mice

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Reduction of axonal caliber does not alleviate motor neuron disease caused by mutant superoxide dismutase 1

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7 January 2021

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Deregulation of Cdk5 in a mouse model of ALS: toxicity alleviated by perikaryal neurofilament inclusions

1 reference

https://api.crossref.org/works/10.1016%2FS0969-9961%2803%2900036-6

7 January 2021

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Distribution of a novel 57 kDa intermediate filament (IF) protein in the nervous system.

1 reference

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7 January 2021

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Transgenic mice expressing an altered murine superoxide dismutase gene provide an animal model of amyotrophic lateral sclerosis

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Apoptotic death of neurons exhibiting peripherin aggregates is mediated by the proinflammatory cytokine tumor necrosis factor-alpha

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7 January 2021

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Mutations in Cu/Zn superoxide dismutase gene are associated with familial amyotrophic lateral sclerosis

1 reference

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7 January 2021

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Increased microglial activation and protein nitration in white matter of the aging monkey☆

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Transgenic mice carrying a human mutant superoxide dismutase transgene develop neuronal cytoskeletal pathology resembling human amyotrophic lateral sclerosis lesions

1 reference

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7 January 2021

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Absence of neurofilaments reduces the selective vulnerability of motor neurons and slows disease caused by a familial amyotrophic lateral sclerosis-linked superoxide dismutase 1 mutant

1 reference

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7 January 2021

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Slowing of axonal transport is a very early event in the toxicity of ALS-linked SOD1 mutants to motor neurons

1 reference

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Differential regulation of peripherin and neurofilament gene expression in regenerating rat DRG neurons

1 reference

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Characterization of neuronal intermediate filament protein expression in cervical spinal motor neurons in sporadic amyotrophic lateral sclerosis (ALS).

1 reference

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An adverse property of a familial ALS-linked SOD1 mutation causes motor neuron disease characterized by vacuolar degeneration of mitochondria

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Identifiers

10.1016/S0969-9961(03)00036-6

1 reference

Europe PubMed Central

PubMed publication ID

3 June 2018

http://europepmc.org/abstract/MED/12828939

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

3 June 2018

http://europepmc.org/abstract/MED/12828939

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