Role of the crosstalk between CTLA-4 and NKG2D in the development of anti-CTLA-4 treatment strategies (Q56897614)

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English	Role of the crosstalk between CTLA-4 and NKG2D in the development of anti-CTLA-4 treatment strategies	article

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instance of

scholarly article

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editorial

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title

Role of the crosstalk between CTLA-4 and NKG2D in the development of anti-CTLA-4 treatment strategies (English)

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author

Sandra Demaria

object named as

Sandra Demaria

series ordinal

1

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Michael L Dustin

object named as

Michael L Dustin

series ordinal

2

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language of work or name

English

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publication date

February 2013

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published in

Immunotherapy

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volume

5

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issue

2

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page(s)

109-12

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cites work

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Suppressing T cell motility induced by anti-CTLA-4 monotherapy improves antitumor effects

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Blockade of CTLA-4 on both effector and regulatory T cell compartments contributes to the antitumor activity of anti-CTLA-4 antibodies

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Anti-CTLA-4 antibody therapy: immune monitoring during clinical development of a novel immunotherapy

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Preoperative CTLA-4 blockade: tolerability and immune monitoring in the setting of a presurgical clinical trial

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The ICOS/ICOSL pathway is required for optimal antitumor responses mediated by anti-CTLA-4 therapy

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Costimulation through NKG2D enhances murine CD8+ CTL function: similarities and differences between NKG2D and CD28 costimulation.

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Cytotoxic T lymphocytes form an antigen-independent ring junction

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Effect of NKG2D ligand expression on host immune responses

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21 January 2018

The DNA damage pathway regulates innate immune system ligands of the NKG2D receptor

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Cell stress-regulated human major histocompatibility complex class I gene expressed in gastrointestinal epithelium

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Targets of protective tumor immunity

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Reversal of the TCR stop signal by CTLA-4

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Immunologic correlates of the abscopal effect in a patient with melanoma

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Tumour-derived soluble MIC ligands impair expression of NKG2D and T-cell activation

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Identifiers

DOI

10.2217/IMT.12.160

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PubMed publication ID

23413900

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