NGF signaling from clathrin-coated vesicles: evidence that signaling endosomes serve as a platform for the Ras-MAPK pathway (Q77334867)

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4 December 2019

title

NGF signaling from clathrin-coated vesicles: evidence that signaling endosomes serve as a platform for the Ras-MAPK pathway (English)

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4 December 2019

1

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4 December 2019

author name string

J S Valletta

series ordinal

2

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4 December 2019

A S Rusnak

series ordinal

3

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4 December 2019

W C Mobley

series ordinal

4

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4 December 2019

publication date

1 December 2001

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4 December 2019

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4 December 2019

volume

32

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4 December 2019

issue

5

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4 December 2019

page(s)

801-814

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NGF signals through TrkA to increase clathrin at the plasma membrane and enhance clathrin-mediated membrane trafficking.

4 December 2019

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Inhibition of clathrin-mediated endocytosis selectively attenuates specific insulin receptor signal transduction pathways

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Endocytosis of activated TrkA: evidence that nerve growth factor induces formation of signaling endosomes

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A signaling organelle containing the nerve growth factor-activated receptor tyrosine kinase, TrkA.

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Retrograde axonal transport of signal transduction proteins in rat sciatic nerve

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Spatially and Functionally Distinct Roles of the PI3-K Effector Pathway during NGF Signaling in Sympathetic Neurons

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Depletion of intracellular potassium arrests coated pit formation and receptor-mediated endocytosis in fibroblasts

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Clathrin-coated vesicles in nervous tissue are involved primarily in synaptic vesicle recycling

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Choline acetyltransferase activity in striatum of neonatal rats increased by nerve growth factor

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7 January 2021

The appendage domain of the AP-2 subunit is not required for assembly or invagination of clathrin-coated pits

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An NGF-TrkA-mediated retrograde signal to transcription factor CREB in sympathetic neurons

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Rapid retrograde tyrosine phosphorylation of trkA and other proteins in rat sympathetic neurons in compartmented cultures

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Cationic amphiphilic drugs inhibit the internalization of cholera toxin to the Golgi apparatus and the subsequent elevation of cyclic AMP.

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Identifiers

DOI

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4 December 2019

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