Complex regulation of CDKs and G1 arrest during the granulocytic differentiation of human myeloblastic leukemia ML-1 cells (Q73074016)

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1 November 2019

title

Complex regulation of CDKs and G1 arrest during the granulocytic differentiation of human myeloblastic leukemia ML-1 cells (English)

1 reference

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1 November 2019

author name string

Shimizu T

series ordinal

1

1 reference

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1 November 2019

Awai N

series ordinal

2

1 reference

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1 November 2019

Takeda K

series ordinal

3

1 reference

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1 November 2019

language of work or name

English

0 references

publication date

1 September 2000

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1 November 2019

1 reference

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1 November 2019

volume

19

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1 November 2019

issue

40

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1 November 2019

page(s)

4640-4646

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Identification of human and mouse p19, a novel CDK4 and CDK6 inhibitor with homology to p16ink4

1 November 2019

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The p21(Cip1) and p27(Kip1) CDK 'inhibitors' are essential activators of cyclin D-dependent kinases in murine fibroblasts

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Evidence that the G1-S and G2-M transitions are controlled by different cdc2 proteins in higher eukaryotes

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Induction of p18INK4c and its predominant association with CDK4 and CDK6 during myogenic differentiation

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Growth suppression by p18, a p16INK4/MTS1- and p14INK4B/MTS2-related CDK6 inhibitor, correlates with wild-type pRb function

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The p21 Cdk-interacting protein Cip1 is a potent inhibitor of G1 cyclin-dependent kinases

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Novel INK4 proteins, p19 and p18, are specific inhibitors of the cyclin D-dependent kinases CDK4 and CDK6

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Induction of cell cycle arrest and B cell terminal differentiation by CDK inhibitor p18(INK4c) and IL-6

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Cdk2-dependent phosphorylation of p27 facilitates its Myc-induced release from cyclin E/cdk2 complexes

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Lovastatin mediated G1 arrest in normal and tumor breast cells is through inhibition of CDK2 activity and redistribution of p21 and p27, independent of p53.

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Kip/Cip and Ink4 Cdk inhibitors cooperate to induce cell cycle arrest in response to TGF-beta

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