BID-dependent and BID-independent pathways for BAX insertion into mitochondria (Q73347784)

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:11139284%20AND%20SRC:MED&resulttype=core&format=json

3 November 2019

title

BID-dependent and BID-independent pathways for BAX insertion into mitochondria (English)

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3 November 2019

6

Korsmeyer SJ

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3 November 2019

author name string

Ruffolo SC

1

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3 November 2019

Breckenridge DG

2

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3 November 2019

Nguyen M

3

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3 November 2019

Goping IS

4

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3 November 2019

Gross A

5

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3 November 2019

Li H

7

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3 November 2019

Yuan J

8

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3 November 2019

Shore GC

9

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3 November 2019

publication date

1 November 2000

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Cell Death & Differentiation

3 November 2019

published in

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3 November 2019

volume

7

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3 November 2019

issue

11

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3 November 2019

page(s)

1101-1108

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BCL-2 family members and the mitochondria in apoptosis

3 November 2019

cites work

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7 January 2021

The Bcl-2 protein family: arbiters of cell survival

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Serine phosphorylation of death agonist BAD in response to survival factor results in binding to 14-3-3 not BCL-X(L)

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7 January 2021

Bid, a Bcl2 interacting protein, mediates cytochrome c release from mitochondria in response to activation of cell surface death receptors

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7 January 2021

Cleavage of BID by caspase 8 mediates the mitochondrial damage in the Fas pathway of apoptosis

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7 January 2021

Caspase cleaved BID targets mitochondria and is required for cytochrome c release, while BCL-XL prevents this release but not tumor necrosis factor-R1/Fas death

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7 January 2021

The proapoptotic activity of the Bcl-2 family member Bim is regulated by interaction with the dynein motor complex

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7 January 2021

Regulated targeting of BAX to mitochondria.

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7 January 2021

Conformation of the Bax C-terminus regulates subcellular location and cell death

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7 January 2021

Mitochondria and apoptosis

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7 January 2021

Prevention of apoptosis by Bcl-2: release of cytochrome c from mitochondria blocked

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7 January 2021

The release of cytochrome c from mitochondria: a primary site for Bcl-2 regulation of apoptosis

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7 January 2021

Cytochrome c and dATP-dependent formation of Apaf-1/caspase-9 complex initiates an apoptotic protease cascade

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7 January 2021

Two CD95 (APO-1/Fas) signaling pathways

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7 January 2021

Caspases induce cytochrome c release from mitochondria by activating cytosolic factors

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7 January 2021

Cytosol-to-membrane redistribution of Bax and Bcl-X(L) during apoptosis

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7 January 2021

Enforced dimerization of BAX results in its translocation, mitochondrial dysfunction and apoptosis

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7 January 2021

Bid-induced conformational change of Bax is responsible for mitochondrial cytochrome c release during apoptosis

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7 January 2021

BAX-induced cell death may not require interleukin 1 beta-converting enzyme-like proteases

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7 January 2021

Bcl-2 prolongs cell survival after Bax-induced release of cytochrome c

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7 January 2021

Mutagenesis of the BH3 domain of BAX identifies residues critical for dimerization and killing

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7 January 2021

Bax-induced cytochrome C release from mitochondria is independent of the permeability transition pore but highly dependent on Mg2+ ions

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7 January 2021

BID: a novel BH3 domain-only death agonist

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7 January 2021

Bax-induced caspase activation and apoptosis via cytochrome c release from mitochondria is inhibitable by Bcl-xL.

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7 January 2021

Bax directly induces release of cytochrome c from isolated mitochondria

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7 January 2021

Involvement of MACH, a novel MORT1/FADD-interacting protease, in Fas/APO-1- and TNF receptor-induced cell death

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7 January 2021

FLICE, a novel FADD-homologous ICE/CED-3-like protease, is recruited to the CD95 (Fas/APO-1) death--inducing signaling complex

1 reference

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7 January 2021

FLICE is activated by association with the CD95 death-inducing signaling complex (DISC).

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7 January 2021

Bcl-x(L) can inhibit apoptosis in cells that have undergone Fas-induced protease activation

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7 January 2021

Targeted disruption of the mouse Caspase 8 gene ablates cell death induction by the TNF receptors, Fas/Apo1, and DR3 and is lethal prenatally.

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7 January 2021

Essential requirement for caspase-8/FLICE in the initiation of the Fas-induced apoptotic cascade

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7 January 2021

Differential modulation of apoptosis sensitivity in CD95 type I and type II cells.

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7 January 2021

Polypeptide and phospholipid composition of the membrane of rat liver peroxisomes: comparison with endoplasmic reticulum and mitochondrial membranes

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7 January 2021

The human mitochondrial import receptor, hTom20p, prevents a cryptic matrix targeting sequence from gaining access to the protein translocation machinery

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7 January 2021

Induction of apoptotic program in cell-free extracts: requirement for dATP and cytochrome c

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7 January 2021

Bid-deficient mice are resistant to Fas-induced hepatocellular apoptosis

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7 January 2021

Withdrawal of IL-7 induces Bax translocation from cytosol to mitochondria through a rise in intracellular pH.

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https://api.crossref.org/works/10.1038%2FSJ.CDD.4400739

7 January 2021

The putative pore-forming domain of Bax regulates mitochondrial localization and interaction with Bcl-X(L).

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https://api.crossref.org/works/10.1038%2FSJ.CDD.4400739

7 January 2021

Bid induces the oligomerization and insertion of Bax into the outer mitochondrial membrane

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7 January 2021

Ion channel activity of the BH3 only Bcl-2 family member, BID.

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7 January 2021

Bax inhibitor-1, a mammalian apoptosis suppressor identified by functional screening in yeast

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7 January 2021

p28 Bap31, a Bcl-2/Bcl-XL- and procaspase-8-associated protein in the endoplasmic reticulum

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7 January 2021

E1A-induced processing of procaspase-8 can occur independently of FADD and is inhibited by Bcl-2

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7 January 2021

Identifiers

DOI

10.1038/SJ.CDD.4400739

1 reference

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:11139284%20AND%20SRC:MED&resulttype=core&format=json

3 November 2019

1 reference