Pages that link to "Q24338423"
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The following pages link to Dopamine covalently modifies and functionally inactivates parkin (Q24338423):
Displaying 50 items.
- Parkin RBR E3 ubiquitin protein ligase (Q2472777) (← links)
- Mussel-inspired surface chemistry for multifunctional coatings (Q22242278) (← links)
- Polydopamine—a nature-inspired polymer coating for biomedical science (Q22250981) (← links)
- PARIS (ZNF746) repression of PGC-1α contributes to neurodegeneration in Parkinson's disease (Q24294809) (← links)
- Parkin mediates neuroprotection through activation of IkappaB kinase/nuclear factor-kappaB signaling (Q24297849) (← links)
- Phosphorylation by the c-Abl protein tyrosine kinase inhibits parkin's ubiquitination and protective function (Q24298958) (← links)
- Parkin interacts with LIM Kinase 1 and reduces its cofilin-phosphorylation activity via ubiquitination (Q24306353) (← links)
- Autoregulation of Parkin activity through its ubiquitin-like domain (Q24307382) (← links)
- The role of oxidative stress in Parkinson's disease (Q24307946) (← links)
- Parkin-dependent degradation of the F-box protein Fbw7β promotes neuronal survival in response to oxidative stress by stabilizing Mcl-1 (Q24308897) (← links)
- Membrane localization of LRRK2 is associated with increased formation of the highly active LRRK2 dimer and changes in its phosphorylation (Q24316270) (← links)
- Structure and Function of Parkin, PINK1, and DJ-1, the Three Musketeers of Neuroprotection (Q24339442) (← links)
- Linking F-box protein 7 and parkin to neuronal degeneration in Parkinson's disease (PD) (Q26752685) (← links)
- Ubiquitin phosphorylation in Parkinson's disease: Implications for pathogenesis and treatment (Q26770342) (← links)
- Parkin Regulation and Neurodegenerative Disorders (Q26770443) (← links)
- Molecular aspects of dopaminergic neurodegeneration: gene-environment interaction in parkin dysfunction (Q26849699) (← links)
- Top-down proteomics in health and disease: challenges and opportunities (Q27002139) (← links)
- Pathologic and therapeutic implications for the cell biology of parkin (Q27010567) (← links)
- Edaravone guards dopamine neurons in a rotenone model for Parkinson's disease (Q27335848) (← links)
- Parkin promotes proteasomal degradation of p62: implication of selective vulnerability of neuronal cells in the pathogenesis of Parkinson's disease (Q27340039) (← links)
- Serotonin 1A Receptors on Astrocytes as a Potential Target for the Treatment of Parkinson's Disease (Q28079379) (← links)
- Glutamate excitotoxicity in neurons triggers mitochondrial and endoplasmic reticulum accumulation of Parkin, and, in the presence of N-acetyl cysteine, mitophagy (Q28387592) (← links)
- Gene-environment interactions: key to unraveling the mystery of Parkinson's disease (Q28388674) (← links)
- Interactions of iron, dopamine and neuromelanin pathways in brain aging and Parkinson's disease (Q28391762) (← links)
- Dopamine and paraquat enhance α-synuclein-induced alterations in membrane conductance (Q28392011) (← links)
- Parkin is protective against proteotoxic stress in a transgenic zebrafish model (Q28474988) (← links)
- Parkin deficiency delays motor decline and disease manifestation in a mouse model of synucleinopathy (Q28475940) (← links)
- Parkin occurs in a stable, non-covalent, approximately 110-kDa complex in brain (Q28505637) (← links)
- Parsing Disease-relevant Protein Modifications from Epiphenomena: Perspective on the Structural Basis of SOD1-Mediated ALS. (Q30101060) (← links)
- The effects of oxidative stress on parkin and other E3 ligases (Q30273796) (← links)
- The Drosophila vesicular monoamine transporter reduces pesticide-induced loss of dopaminergic neurons (Q30430534) (← links)
- MPTP and DSP-4 susceptibility of substantia nigra and locus coeruleus catecholaminergic neurons in mice is independent of parkin activity (Q30502685) (← links)
- Parthanatos mediates AIMP2-activated age-dependent dopaminergic neuronal loss (Q30547326) (← links)
- Synthesis and structural characterization of soluble neuromelanin analogs provides important clues to its biosynthesis (Q30575408) (← links)
- Aldehyde dehydrogenase 1 defines and protects a nigrostriatal dopaminergic neuron subpopulation (Q30581658) (← links)
- Strategies for stabilizing superoxide dismutase (SOD1), the protein destabilized in the most common form of familial amyotrophic lateral sclerosis (Q30992306) (← links)
- Chemistry of periodate-mediated cross-linking of 3,4-dihydroxylphenylalanine-containing molecules to proteins (Q33264179) (← links)
- Role of the ubiquitin proteasome system in Parkinson's disease (Q33307874) (← links)
- Dopamine quinone modifies and decreases the abundance of the mitochondrial selenoprotein glutathione peroxidase 4. (Q33705939) (← links)
- Inhibition of apoptotic Bax translocation to the mitochondria is a central function of parkin. (Q34007710) (← links)
- Identification and characterization of a novel endogenous murine parkin mutation (Q34012526) (← links)
- Protein kinases CK1 and CK2 as new targets for neurodegenerative diseases (Q34122676) (← links)
- Small, N-terminal tags activate Parkin E3 ubiquitin ligase activity by disrupting its autoinhibited conformation. (Q34230329) (← links)
- Human SOD2 modification by dopamine quinones affects enzymatic activity by promoting its aggregation: possible implications for Parkinson's disease (Q34313803) (← links)
- Genome-wide association study of monoamine metabolite levels in human cerebrospinal fluid. (Q34322551) (← links)
- Role of oxidative stress in Parkinson's disease (Q34339252) (← links)
- Novel regulation of parkin function through c-Abl-mediated tyrosine phosphorylation: implications for Parkinson's disease (Q34577087) (← links)
- Dopamine-mediated oxidation of methionine 127 in α-synuclein causes cytotoxicity and oligomerization of α-synuclein (Q34606278) (← links)
- Mitochondrial morphogenesis, distribution, and Parkinson disease: insights from PINK1 (Q34610341) (← links)
- Behavioral phenotyping of Parkin-deficient mice: looking for early preclinical features of Parkinson's disease (Q34654923) (← links)