Bax can antagonize Bcl-XL during etoposide and cisplatin-induced cell death independently of its heterodimerization with Bcl-XL (Q28587967)

31 January 2017

title

Bax can antagonize Bcl-XL during etoposide and cisplatin-induced cell death independently of its heterodimerization with Bcl-XL (English)

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BCL2-associated X protein

31 January 2017

main subject

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GOA release 2020-03-11

BCL2-like 1

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protein heterodimerization activity

GOA release 2020-03-11

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P. L. Simonian

1

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31 January 2017

D. A. Grillot

series ordinal

2

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31 January 2017

R. Merino

series ordinal

3

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31 January 2017

G. Nuñez

series ordinal

4

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31 January 2017

language of work or name

English

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publication date

13 September 1996

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Journal of Biological Chemistry

31 January 2017

published in

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31 January 2017

volume

271

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31 January 2017

issue

37

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31 January 2017

page(s)

22764–22772

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Social controls on cell survival and cell death

31 January 2017

cites work

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The t(14;18) chromosome translocations involved in B-cell neoplasms result from mistakes in VDJ joining

21 January 2018

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Bcl-2 gene promotes haemopoietic cell survival and cooperates with c-myc to immortalize pre-B cells

21 January 2018

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BH1 and BH2 domains of Bcl-2 are required for inhibition of apoptosis and heterodimerization with Bax.

21 January 2018

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bcl-x, a bcl-2-related gene that functions as a dominant regulator of apoptotic cell death

21 January 2018

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Bcl-2 heterodimerizes in vivo with a conserved homolog, Bax, that accelerates programmed cell death

21 January 2018

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Bax-independent inhibition of apoptosis by Bcl-XL.

21 January 2018

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Induction of apoptosis by the Bcl-2 homologue Bak

21 January 2018

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Cloning of a bcl-2 homologue by interaction with adenovirus E1B 19K

21 January 2018

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Modulation of apoptosis by the widely distributed Bcl-2 homologue Bak

21 January 2018

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CD28 costimulation can promote T cell survival by enhancing the expression of Bcl-XL.

21 January 2018

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A rapid and simple method for measuring thymocyte apoptosis by propidium iodide staining and flow cytometry

21 January 2018

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The E1B 19K protein blocks apoptosis by interacting with and inhibiting the p53-inducible and death-promoting Bax protein

21 January 2018

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21 January 2018

Identifiers

DOI

10.1074/JBC.271.37.22764

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