Superoxide dismutase 1 and tgSOD1 mouse spinal cord seed fibrils, suggesting a propagative cell death mechanism in amyotrophic lateral sclerosis (Q21136374)

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Superoxide Dismutase 1 and tgSOD1G93A Mouse Spinal Cord Seed Fibrils, Suggesting a Propagative Cell Death Mechanism in Amyotrophic Lateral Sclerosis

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English	Superoxide dismutase 1 and tgSOD1 mouse spinal cord seed fibrils, suggesting a propagative cell death mechanism in amyotrophic lateral sclerosis	scientific article	Superoxide Dismutase 1 and tgSOD1G93A Mouse Spinal Cord Seed Fibrils, Suggesting a Propagative Cell Death Mechanism in Amyotrophic Lateral Sclerosis

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scholarly article

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Superoxide dismutase 1 and tgSOD1 mouse spinal cord seed fibrils, suggesting a propagative cell death mechanism in amyotrophic lateral sclerosis (English)

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amyotrophic lateral sclerosis

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1 reference

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inferred from title

Graham S Jackson

object named as

Graham S Jackson

6

author given names

Graham S

author last names

Jackson

1 reference

PubMed publication ID

17 August 2022

Elizabeth M. C. Fisher

5

object named as

Elizabeth M C Fisher

author given names

Elizabeth M C

author last names

Fisher

1 reference

PubMed publication ID

17 August 2022

4

author given names

John

author last names

Collinge

object named as

John Collinge

1 reference

PubMed publication ID

17 August 2022

author name string

Ruth Chia

1

author given names

Ruth

author last names

Chia

1 reference

PubMed publication ID

17 August 2022

M Howard Tattum

2

author given names

M Howard

author last names

Tattum

1 reference

PubMed publication ID

17 August 2022

Samantha Jones

3

author given names

Samantha

author last names

Jones

1 reference

PubMed publication ID

17 August 2022

language of work or name

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publication date

2010

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5

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5

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e10627

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copyright license

Creative Commons Attribution 4.0 International

13 May 2010

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April 2022 Public Data File from Crossref

copyright status

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SOD1 and amyotrophic lateral sclerosis: mutations and oligomerization

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Non-cell autonomous effect of glia on motor neurons in an embryonic stem cell-based ALS model

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19 March 2017

Chromogranin-mediated secretion of mutant superoxide dismutase proteins linked to amyotrophic lateral sclerosis

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https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2869360

19 March 2017

Mutations in the endosomal ESCRTIII-complex subunit CHMP2B in frontotemporal dementia

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https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2869360

19 March 2017

ALS mutants of human superoxide dismutase form fibrous aggregates via framework destabilization

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https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2869360

19 March 2017

Amyloid-like filaments and water-filled nanotubes formed by SOD1 mutant proteins linked to familial ALS

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https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2869360

19 March 2017

Amyotrophic lateral sclerosis and structural defects in Cu,Zn superoxide dismutase

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https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2869360

19 March 2017

Mutations in Cu/Zn superoxide dismutase gene are associated with familial amyotrophic lateral sclerosis

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https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2869360

19 March 2017

Modification of superoxide dismutase 1 (SOD1) properties by a GFP tag--implications for research into amyotrophic lateral sclerosis (ALS)

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19 March 2017

A general model of prion strains and their pathogenicity

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https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2869360

19 March 2017

Superoxide dismutase multigene family: a comparison of the CuZn-SOD (SOD1), Mn-SOD (SOD2), and EC-SOD (SOD3) gene structures, evolution, and expression

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https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2869360

19 March 2017

Formation of neuronal intranuclear inclusions underlies the neurological dysfunction in mice transgenic for the HD mutation

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https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2869360

19 March 2017

Mice devoid of PrP are resistant to scrapie

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https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2869360

19 March 2017

Evidence of calcium- and SNARE-dependent release of CuZn superoxide dismutase from rat pituitary GH3 cells and synaptosomes in response to depolarization

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https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2869360

19 March 2017

Impaired extracellular secretion of mutant superoxide dismutase 1 associates with neurotoxicity in familial amyotrophic lateral sclerosis

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https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2869360

19 March 2017

Motor neuron degeneration in mice that express a human Cu,Zn superoxide dismutase mutation

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https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2869360

7 April 2017

ALS: a disease of motor neurons and their nonneuronal neighbors

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https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2869360

7 April 2017

Prion-like mechanisms in neurodegenerative diseases

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https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2869360

12 September 2017

Genetics of motor neuron disorders: new insights into pathogenic mechanisms

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https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2869360

12 September 2017

Variation in aggregation propensities among ALS-associated variants of SOD1: correlation to human disease.

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https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2869360

12 September 2017

Loss of metal ions, disulfide reduction and mutations related to familial ALS promote formation of amyloid-like aggregates from superoxide dismutase

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2869360

12 September 2017

Initiation and elongation in fibrillation of ALS-linked superoxide dismutase

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2869360

12 September 2017

Detergent-insoluble aggregates associated with amyotrophic lateral sclerosis in transgenic mice contain primarily full-length, unmodified superoxide dismutase-1.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2869360

12 September 2017

Amyotrophic lateral sclerosis models and human neuropathology: similarities and differences

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2869360

12 September 2017

Soluble misfolded subfractions of mutant superoxide dismutase-1s are enriched in spinal cords throughout life in murine ALS models

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2869360

12 September 2017

Metal-free superoxide dismutase forms soluble oligomers under physiological conditions: a possible general mechanism for familial ALS

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2869360

12 September 2017

How do ALS-associated mutations in superoxide dismutase 1 promote aggregation of the protein?

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2869360

12 September 2017

Mutant SOD1 instability: implications for toxicity in amyotrophic lateral sclerosis

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https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2869360

12 September 2017

Genetics of familial and sporadic amyotrophic lateral sclerosis

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2869360

12 September 2017

Rational targeting for prion therapeutics.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2869360

12 September 2017

Cu/Zn superoxide dismutase mutants associated with amyotrophic lateral sclerosis show enhanced formation of aggregates in vitro

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2869360

12 September 2017

Self-assembly of polyglutamine-containing huntingtin fragments into amyloid-like fibrils: implications for Huntington's disease pathology

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2869360

12 September 2017

Effect of weak bases on the intralysosomal pH in mouse peritoneal macrophages

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2869360

12 September 2017

Complete loss of post-translational modifications triggers fibrillar aggregation of SOD1 in the familial form of amyotrophic lateral sclerosis

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2869360

27 September 2017

Evidence for secretion of Cu,Zn superoxide dismutase via exosomes from a cell model of amyotrophic lateral sclerosis

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2869360

27 September 2017

Prion protein (PrP) with amino-proximal deletions restoring susceptibility of PrP knockout mice to scrapie.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2869360

27 September 2017

Architecture and anatomy of the chromosomal locus in human chromosome 21 encoding the Cu/Zn superoxide dismutase.

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27 September 2017

Conformational diversity of wild-type Tau fibrils specified by templated conformation change

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https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2869360

30 May 2018

Oxidation versus aggregation - how do SOD1 mutants cause ALS?

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2869360

30 May 2018

Amyotrophic lateral sclerosis: current issues in classification, pathogenesis and molecular pathology

1 reference

https://api.crossref.org/works/10.1371%2FJOURNAL.PONE.0010627

21 January 2018

Focality of upper and lower motor neuron degeneration at the clinical onset of ALS.

1 reference

https://api.crossref.org/works/10.1371%2FJOURNAL.PONE.0010627

21 January 2018

Implications of ALS focality: rostral-caudal distribution of lower motor neuron loss postmortem

1 reference

https://api.crossref.org/works/10.1371%2FJOURNAL.PONE.0010627

21 January 2018

The phagocytic capacity of neurones

1 reference

https://api.crossref.org/works/10.1371%2FJOURNAL.PONE.0010627

21 January 2018

Amyotrophic lateral sclerosis mutations have the greatest destabilizing effect on the apo- and reduced form of SOD1, leading to unfolding and oxidative aggregation.

1 reference

https://api.crossref.org/works/10.1371%2FJOURNAL.PONE.0010627

21 January 2018

Rapid disease progression correlates with instability of mutant SOD1 in familial ALS

1 reference

https://api.crossref.org/works/10.1371%2FJOURNAL.PONE.0010627

21 January 2018

Fibrillar inclusions and motor neuron degeneration in transgenic mice expressing superoxide dismutase 1 with a disrupted copper-binding site

1 reference

https://api.crossref.org/works/10.1371%2FJOURNAL.PONE.0010627

21 January 2018

Prefibrillar amyloid aggregates could be generic toxins in higher organisms.

1 reference

https://api.crossref.org/works/10.1371%2FJOURNAL.PONE.0010627

21 January 2018

Wild-Type Nonneuronal Cells Extend Survival of SOD1 Mutant Motor Neurons in ALS Mice

1 reference

https://api.crossref.org/works/10.1371%2FJOURNAL.PONE.0010627

21 January 2018

Probing the mechanism of insulin fibril formation with insulin mutants

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https://api.crossref.org/works/10.1371%2FJOURNAL.PONE.0010627

21 January 2018

Intranuclear inclusions and neuritic aggregates in transgenic mice expressing a mutant N-terminal fragment of huntingtin

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2869360

29 November 2018

Disulphide-reduced superoxide dismutase-1 in CNS of transgenic amyotrophic lateral sclerosis models

1 reference

https://pubmed.ncbi.nlm.nih.gov/20498711

12 December 2020

based on heuristic

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Insoluble Mutant SOD1 Is Partly Oligoubiquitinated in Amyotrophic Lateral Sclerosis Mice

1 reference

https://pubmed.ncbi.nlm.nih.gov/20498711

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Stabilization of mutant Cu/Zn superoxide dismutase (SOD1) protein by coexpressed wild SOD1 protein accelerates the disease progression in familial amyotrophic lateral sclerosis mice

1 reference

https://pubmed.ncbi.nlm.nih.gov/20498711

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Disease penetrance in amyotrophic lateral sclerosis associated with mutations in the SOD1 gene

1 reference

https://pubmed.ncbi.nlm.nih.gov/20498711

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Cu/Zn superoxide dismutase-like immunoreactivity in Lewy body-like inclusions of sporadic amyotrophic lateral sclerosis

1 reference

https://pubmed.ncbi.nlm.nih.gov/20498711

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Sporadic amyotrophic lateral sclerosis with dementia and Cu/Zn superoxide dismutase-positive Lewy body-like inclusions

1 reference

https://pubmed.ncbi.nlm.nih.gov/20498711

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

SOD1 aggregates in ALS: cause, correlate or consequence?

1 reference

https://pubmed.ncbi.nlm.nih.gov/20498711

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Wild-type superoxide dismutase acquires binding and toxic properties of ALS-linked mutant forms through oxidation

1 reference

https://pubmed.ncbi.nlm.nih.gov/20498711

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Neuron-specific expression of mutant superoxide dismutase is sufficient to induce amyotrophic lateral sclerosis in transgenic mice

1 reference

https://pubmed.ncbi.nlm.nih.gov/20498711

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Identifiers

10.1371/JOURNAL.PONE.0010627

1 reference

Consolidated OpenCitations Corpus – April 2017

OpenCitations bibliographic resource ID

2010PLoSO...510627C

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OpenCitations bibliographic resource ID

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Consolidated OpenCitations Corpus – April 2017

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