Functional roles and therapeutic targeting of gelatinase B and chemokines in multiple sclerosis (Q35592638)

9 August 2017

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title

Functional roles and therapeutic targeting of gelatinase B and chemokines in multiple sclerosis (English)

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9 August 2017

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1

Ghislain Opdenakker

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author name string

Inge Nelissen

series ordinal

2

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9 August 2017

Jo Van Damme

series ordinal

3

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9 August 2017

publication date

1 December 2003

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9 August 2017

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9 August 2017

volume

2

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9 August 2017

issue

12

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9 August 2017

page(s)

747-756

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A full genome search in multiple sclerosis

9 August 2017

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Polymorphism analysis suggests that the gelatinase B gene is not a susceptibility factor for multiple sclerosis

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Gelatinase B: a tuner and amplifier of immune functions.

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Biochemistry and molecular biology of gelatinase B or matrix metalloproteinase-9 (MMP-9)

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Evaluation of gelatinases and IL-6 in the cerebrospinal fluid of patients with optic neuritis, multiple sclerosis and other inflammatory neurological diseases

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Matrix metalloproteinases in the normal human central nervous system, microglial nodules, and multiple sclerosis lesions

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The Expression of Tissue-type Plasminogen Activator, Matrix Metalloproteases and Endogenous Inhibitors in the Central Nervous System in Multiple Sclerosis

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Enhanced expression of MMP-7 and MMP-9 in demyelinating multiple sclerosis lesions

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Matrix metalloproteinase-12 is expressed in phagocytotic macrophages in active multiple sclerosis lesions

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Matrix metalloproteinase-9 (gelatinase B) is selectively elevated in CSF during relapses and stable phases of multiple sclerosis

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Serum gelatinase B, TIMP-1 and TIMP-2 levels in multiple sclerosis. A longitudinal clinical and MRI study

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Matrix metalloproteinases and their tissue inhibitors as markers of disease subtype and response to interferon-beta therapy in relapsing and secondary-progressive multiple sclerosis patients

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Monocyte-derived dendritic cells express and secrete matrix-degrading metalloproteinases and their inhibitors and are imbalanced in multiple sclerosis

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Serum MMP-2 and MMP-9 are elevated in different multiple sclerosis subtypes

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The expression profile of matrix metalloproteinases (MMPs) and their inhibitors (TIMPs) in lesions and normal appearing white matter of multiple sclerosis

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7 January 2021

Gelatinase B is present in the cerebrospinal fluid during experimental autoimmune encephalomyelitis and cleaves myelin basic protein.

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Matrix metalloproteinase expression in an experimentally-induced DTH model of multiple sclerosis in the rat CNS.

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Matrix metalloproteinase-9 and -7 are regulated in experimental autoimmune encephalomyelitis

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Up-regulation of MMP-8 and MMP-9 activity in the BALB/c mouse spinal cord correlates with the severity of experimental autoimmune encephalomyelitis

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7 January 2021

The cysteine switch: a principle of regulation of metalloproteinase activity with potential applicability to the entire matrix metalloproteinase gene family

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S-nitrosylation of matrix metalloproteinases: signaling pathway to neuronal cell death

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Expression of matrix metalloproteinase-9 and its inhibitors in mononuclear blood cells of patients with multiple sclerosis

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Interferon beta-1b inhibits gelatinase secretion and in vitro migration of human T cells: a possible mechanism for treatment efficacy in multiple sclerosis

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Migratory behavior of lymphocytes isolated from multiple sclerosis patients: effects of interferon beta-1b therapy

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Human Monocyte-Derived Dendritic Cells Produce Bioactive Gelatinase B: Inhibition by IFN-β

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The gelatinase inhibitory activity of tetracyclines and chemically modified tetracycline analogues as measured by a novel microtiter assay for inhibitors.

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Treatment of relapsing paralysis in experimental encephalomyelitis by targeting Th1 cells through atorvastatin

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Fibronectin-mediated cell adhesion is required for induction of 92-kDa type IV collagenase/gelatinase (MMP-9) gene expression during macrophage differentiation. The signaling role of protein kinase C-beta

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Chemokine expression in GKO mice (lacking interferon-gamma) with experimental autoimmune encephalomyelitis

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7 January 2021

Role of chemokines, neuronal projections, and the blood-brain barrier in the enhancement of cerebral EAE following focal brain damage

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7 January 2021

Expression of the beta-chemokine receptors CCR2, CCR3 and CCR5 in multiple sclerosis central nervous system tissue

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7 January 2021

Chemokines and chemokine receptors in inflammation of the nervous system: manifold roles and exquisite regulation.

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Chemokine expression in murine experimental allergic encephalomyelitis

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7 January 2021

10.1016/S1474-4422(03)00587-8

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DOI

1 reference

9 August 2017

1 reference