Caspase-8 is required for cell death induced by expanded polyglutamine repeats. (Q40961806)

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4 December 2019

title

Caspase-8 is required for cell death induced by expanded polyglutamine repeats (English)

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4 December 2019

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1

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4 December 2019

author name string

C J Xu

2

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4 December 2019

P Juo

3

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4 December 2019

A Kakizaka

4

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4 December 2019

J Blenis

5

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4 December 2019

J Yuan

6

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4 December 2019

language of work or name

English

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publication date

1 March 1999

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4 December 2019

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4 December 2019

volume

22

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4 December 2019

issue

3

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4 December 2019

page(s)

623-633

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The proto-oncogene bcl-2 can selectively rescue neurotrophic factor-dependent neurons from apoptosis

4 December 2019

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FADD/MORT1 is a common mediator of CD95 (Fas/APO-1) and tumor necrosis factor receptor-induced apoptosis

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Cleavage of huntingtin by apopain, a proapoptotic cysteine protease, is modulated by the polyglutamine tract

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A novel gene containing a trinucleotide repeat that is expanded and unstable on Huntington's disease chromosomes.

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Suppression of aggregate formation and apoptosis by transglutaminase inhibitors in cells expressing truncated DRPLA protein with an expanded polyglutamine stretch.

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Expanded polyglutamine in the Machado-Joseph disease protein induces cell death in vitro and in vivo

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Caspase-3 is required for alpha-fodrin cleavage but dispensable for cleavage of other death substrates in apoptosis

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CAG expansions in a novel gene for Machado-Joseph disease at chromosome 14q32.1

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Ataxin-1 nuclear localization and aggregation: role in polyglutamine-induced disease in SCA1 transgenic mice

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7 January 2021

Cleavage of BID by caspase 8 mediates the mitochondrial damage in the Fas pathway of apoptosis

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Identifiers

DOI

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4 December 2019

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