Bax translocation to mitochondria subsequent to a rapid loss of mitochondrial membrane potential (Q43722380)

26 November 2017

title

Bax translocation to mitochondria subsequent to a rapid loss of mitochondrial membrane potential (English)

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26 November 2017

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Smaili SS

1

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26 November 2017

Hsu YT

2

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26 November 2017

Sanders KM

3

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26 November 2017

Russell JT

4

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26 November 2017

Youle RJ

5

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26 November 2017

publication date

1 September 2001

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Cell Death & Differentiation

26 November 2017

published in

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26 November 2017

volume

8

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26 November 2017

issue

9

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26 November 2017

page(s)

909-920

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Bcl-2 heterodimerizes in vivo with a conserved homolog, Bax, that accelerates programmed cell death

26 November 2017

cites work

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400889

Bax-deficient mice with lymphoid hyperplasia and male germ cell death

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400889

Bax-induced caspase activation and apoptosis via cytochrome c release from mitochondria is inhibitable by Bcl-xL.

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400889

Structure-function comparisons of the proapoptotic protein Bax in yeast and mammalian cells

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400889

BH1 and BH2 domains of Bcl-2 are required for inhibition of apoptosis and heterodimerization with Bax.

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400889

Movement of Bax from the cytosol to mitochondria during apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400889

Cytosol-to-membrane redistribution of Bax and Bcl-X(L) during apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400889

Regulated targeting of BAX to mitochondria.

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400889

Enforced dimerization of BAX results in its translocation, mitochondrial dysfunction and apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400889

Conformation of the Bax C-terminus regulates subcellular location and cell death

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400889

Biochemical and genetic analysis of the mitochondrial response of yeast to BAX and BCL-X(L)

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400889

Sequential reduction of mitochondrial transmembrane potential and generation of reactive oxygen species in early programmed cell death

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400889

Induction of apoptotic program in cell-free extracts: requirement for dATP and cytochrome c

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400889

Bcl-2 inhibits the mitochondrial release of an apoptogenic protease

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400889

Molecular characterization of mitochondrial apoptosis-inducing factor

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400889

Mitochondrial control of nuclear apoptosis.

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400889

Mitochondrial permeability transition is a central coordinating event of apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400889

Mitochondrial depolarization accompanies cytochrome c release during apoptosis in PC6 cells

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400889

The mitochondrial death/life regulator in apoptosis and necrosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400889

Mitochondrial cytochrome c release in apoptosis occurs upstream of DEVD-specific caspase activation and independently of mitochondrial transmembrane depolarization.

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400889

The coordinate release of cytochrome c during apoptosis is rapid, complete and kinetically invariant

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400889

Bax directly induces release of cytochrome c from isolated mitochondria

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400889

The permeability transition pore complex: a target for apoptosis regulation by caspases and bcl-2-related proteins

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400889

Mitochondria in Ca2+ signaling and apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400889

The mitochondrial permeability transition.

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400889

Bax-induced cytochrome C release from mitochondria is independent of the permeability transition pore but highly dependent on Mg2+ ions

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400889

Cytochrome c release from brain mitochondria is independent of the mitochondrial permeability transition.

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400889

Inhibition of Bax channel-forming activity by Bcl-2

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400889

Bax oligomerization is required for channel-forming activity in liposomes and to trigger cytochrome c release from mitochondria

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400889

Bax, but not Bcl-xL, decreases the lifetime of planar phospholipid bilayer membranes at subnanomolar concentrations

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400889

The putative pore-forming domain of Bax regulates mitochondrial localization and interaction with Bcl-X(L).

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400889

Channel formation by antiapoptotic protein Bcl-2.

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400889

Nitric oxide donors, nitrosothiols and mitochondrial respiration inhibitors induce caspase activation by different mechanisms

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400889

The BH3 domain is required for caspase-independent cell death induced by Bax and oligomycin

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400889

Inhibition of mitochondrial function in HL60 cells is associated with an increased apoptosis and expression of CD14

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400889

Mitochondrial respiratory chain inhibitors induce apoptosis.

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400889

The Mitochondrial F0F1-ATPase proton pump is required for function of the proapoptotic protein Bax in yeast and mammalian cells.

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400889

Intracellular adenosine triphosphate (ATP) concentration: a switch in the decision between apoptosis and necrosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400889

Role of oxidative phosphorylation in Bax toxicity.

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400889

Withdrawal of IL-7 induces Bax translocation from cytosol to mitochondria through a rise in intracellular pH.

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400889

Changes in intramitochondrial and cytosolic pH: early events that modulate caspase activation during apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400889

Simultaneous imaging of cell and mitochondrial membrane potentials

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400889

Physiological cytosolic Ca2+ transients evoke concurrent mitochondrial depolarizations.

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400889

Mitochondria and cell death. Mechanistic aspects and methodological issues

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400889

Bax and adenine nucleotide translocator cooperate in the mitochondrial control of apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400889

BAX-induced cell death may not require interleukin 1 beta-converting enzyme-like proteases

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400889

Mitochondria and neuronal survival

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400889

Prevention of apoptosis by Bcl-2: release of cytochrome c from mitochondria blocked

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400889

Investigation of bax-induced release of cytochrome c from yeast mitochondria permeability of mitochondrial membranes, role of VDAC and ATP requirement

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400889

Peg3/Pw1 promotes p53-mediated apoptosis by inducing Bax translocation from cytosol to mitochondria

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400889

Posttranslational N-myristoylation of BID as a molecular switch for targeting mitochondria and apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400889

Elevation of resting mitochondrial membrane potential of neural cells by cyclosporin A, BAPTA-AM, and bcl-2.

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400889

Nonlinear propagation of agonist-induced cytoplasmic calcium waves in single astrocytes.

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400889

Widespread elimination of naturally occurring neuronal death in Bax-deficient mice

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400889

7 January 2021

Caspase-dependent apoptosis of COS-7 cells induced by Bax overexpression: differential effects of Bcl-2 and Bcl-xL on Bax-induced caspase activation and apoptosis

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400889

7 January 2021

The biochemistry of programmed cell death

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400889

7 January 2021

Mitochondrial perturbations define lymphocytes undergoing apoptotic depletion in vivo

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400889

7 January 2021

The overexpression of Bax produces cell death upon induction of the mitochondrial permeability transition

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400889

7 January 2021

Nonionic detergents induce dimerization among members of the Bcl-2 family

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400889

7 January 2021

Cardiolipin provides specificity for targeting of tBid to mitochondria

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400889

7 January 2021

BID-dependent and BID-independent pathways for BAX insertion into mitochondria

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400889

7 January 2021

Bid-induced cytochrome c release is mediated by a pathway independent of mitochondrial permeability transition pore and Bax.

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400889

7 January 2021

Permeability transition pore regulates both mitochondrial membrane potential and agonist-evoked Ca2+ signals in oligodendrocyte progenitors

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400889

7 January 2021

Identifiers

DOI

10.1038/SJ.CDD.4400889

1 reference

Dimensions Publication ID

26 November 2017

1007591687

0 references

1 reference