Tissue distribution of protease resistant prion protein in variant Creutzfeldt-Jakob disease using a highly sensitive immunoblotting assay (Q57092938)

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9 November 2019

title

Tissue distribution of protease resistant prion protein in variant Creutzfeldt-Jakob disease using a highly sensitive immunoblotting assay (English)

1 reference

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Creutzfeldt-Jakob disease

9 November 2019

main subject

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1

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9 November 2019

Andrew F. Hill

3

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9 November 2019

Philip J. Luthert

6

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9 November 2019

author name string

S Joiner

2

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9 November 2019

M Desbruslais

5

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9 November 2019

J Collinge

7

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9 November 2019

T A Campbell

4

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9 November 2019

language of work or name

English

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publication date

1 July 2001

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9 November 2019

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9 November 2019

volume

358

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9 November 2019

issue

9277

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9 November 2019

page(s)

171-180

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Self-replication and scrapie

9 November 2019

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Biochemical and physical properties of the prion protein from two strains of the transmissible mink encephalopathy agent

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Molecular analysis of prion strain variation and the aetiology of 'new variant' CJD.

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Evidence for the conformation of the pathologic isoform of the prion protein enciphering and propagating prion diversity

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Eight prion strains have PrP(Sc) molecules with different conformations

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Strain-specific prion-protein conformation determined by metal ions

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The same prion strain causes vCJD and BSE

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Transmissions to mice indicate that 'new variant' CJD is caused by the BSE agent

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Variant Creutzfeldt-Jakob disease.

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Diagnosis of new variant Creutzfeldt-Jakob disease by tonsil biopsy

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Investigation of variant Creutzfeldt-Jakob disease and other human prion diseases with tonsil biopsy samples.

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Retrospective study of prion-protein accumulation in tonsil and appendix tissues.

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7 January 2021

Creutzfeldt-Jakob disease and blood transfusion

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A crucial role for B cells in neuroinvasive scrapie.

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Blood transfusion risk: protecting against the unknown

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Danger of accidental person-to-person transmission of Creutzfeldt-Jakob disease by surgery

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7 January 2021

Surgical treatment and risk of sporadic Creutzfeldt-Jakob disease: a case-control study

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Unaltered susceptibility to BSE in transgenic mice expressing human prion protein.

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Preliminary observations on the pathogenesis of experimental bovine spongiform encephalopathy (BSE): an update

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Limited detection of sternal bone marrow infectivity in the clinical phase of experimental bovine spongiform encephalopathy (BSE).

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Transmission of bovine spongiform encephalopathy and scrapie to mice: strain variation and the species barrier

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Compelling transgenetic evidence for transmission of bovine spongiform encephalopathy prions to humans

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7 January 2021

Tissue handling in suspected Creutzfeldt-Jakob disease (CJD) and other human spongiform encephalopathies (prion diseases)

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7 January 2021

Monitoring plasma processing steps with a sensitive Western blot assay for the detection of the prion protein

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7 January 2021

Further studies of blood infectivity in an experimental model of transmissible spongiform encephalopathy, with an explanation of why blood components do not transmit Creutzfeldt-Jakob disease in humans.

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7 January 2021

Mice devoid of PrP are resistant to scrapie

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7 January 2021

Species-barrier-independent prion replication in apparently resistant species

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7 January 2021

Prion immunoreactivity in appendix before clinical onset of variant Creutzfeldt-Jakob disease

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7 January 2021

Predicted vCJD mortality in Great Britain

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7 January 2021

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7 January 2021

Infectivity in extraneural tissues following intraocular scrapie infection

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7 January 2021

Measurement of the scrapie agent using an incubation time interval assay

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7 January 2021

Prion protein devoid of the octapeptide repeat region restores susceptibility to scrapie in PrP knockout mice.

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7 January 2021

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Fatal familial insomnia: a second kindred with mutation of prion protein gene at codon 178

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Identifiers

DOI

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https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:11476832%20AND%20SRC:MED&resulttype=core&format=json

9 November 2019

1 reference