Pages that link to "Q52457566"
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The following pages link to Redox cycling of anthracyclines by cardiac mitochondria. II. Formation of superoxide anion, hydrogen peroxide, and hydroxyl radical. (Q52457566):
Displaying 50 items.
- Fatty acid amide hydrolase is a key regulator of endocannabinoid-induced myocardial tissue injury (Q24595633) (← links)
- Pharmacological inhibition of CB1 cannabinoid receptor protects against doxorubicin-induced cardiotoxicity (Q24648835) (← links)
- Mitochondrial toxin 3-nitropropionic acid induces cardiac and neurotoxicity differentially in mice (Q28360954) (← links)
- Evidence suggesting a role for hydroxyl radical in gentamicin-induced acute renal failure in rats (Q28360981) (← links)
- Doxorubicin in vivo rapidly alters expression and translation of myocardial electron transport chain genes, leads to ATP loss and caspase 3 activation (Q28383334) (← links)
- A switching mechanism in doxorubicin bioactivation can be exploited to control doxorubicin toxicity (Q28477095) (← links)
- ROCK1 deficiency enhances protective effects of antioxidants against apoptosis and cell detachment (Q28540406) (← links)
- Dissecting the Mechanisms of Doxorubicin and Oxidative Stress-Induced Cytotoxicity: The Involvement of Actin Cytoskeleton and ROCK1 (Q28546141) (← links)
- Differential regulation of NF-kappaB activation and function by topoisomerase II inhibitors (Q33240665) (← links)
- A mouse model for juvenile doxorubicin-induced cardiac dysfunction (Q33572418) (← links)
- Iscador Qu inhibits doxorubicin-induced senescence of MCF7 cells (Q33812688) (← links)
- Dietary nitrate supplementation protects against Doxorubicin-induced cardiomyopathy by improving mitochondrial function. (Q34022675) (← links)
- NAD(P)H:quinone oxidoreductase 1 is induced by progesterone in cardiomyocytes (Q34025654) (← links)
- Inhibition of apoptosis by progesterone in cardiomyocytes (Q34047552) (← links)
- Long-acting phosphodiesterase-5 inhibitor tadalafil attenuates doxorubicin-induced cardiomyopathy without interfering with chemotherapeutic effect. (Q34128868) (← links)
- Dexrazoxane for the prevention of cardiac toxicity and treatment of extravasation injury from the anthracycline antibiotics (Q34255916) (← links)
- Anthracycline cardiotoxicity: prevalence, pathogenesis and treatment (Q34639876) (← links)
- Reversing mitochondrial dysfunction, fatigue and the adverse effects of chemotherapy of metastatic disease by molecular replacement therapy (Q34721743) (← links)
- Attenuation of Doxorubicin-induced Cardiotoxicity by Tadalafil: A Long Acting Phosphodiesterase-5 Inhibitor. (Q34793299) (← links)
- Activation of the aryl hydrocarbon receptor by doxorubicin mediates cytoprotective effects in the heart (Q34808490) (← links)
- Chemotherapy-induced weakness and fatigue in skeletal muscle: the role of oxidative stress (Q35222202) (← links)
- Apoptosis in Anthracycline Cardiomyopathy (Q35642302) (← links)
- Cannabidiol Protects against Doxorubicin-Induced Cardiomyopathy by Modulating Mitochondrial Function and Biogenesis (Q35707221) (← links)
- Induction of antioxidant and detoxification response by oxidants in cardiomyocytes: evidence from gene expression profiling and activation of Nrf2 transcription factor (Q35757988) (← links)
- Mesenchymal stem cell therapy for doxorubicin cardiomyopathy: hopes and fears (Q35774310) (← links)
- Analysis of proteome changes in doxorubicin-treated adult rat cardiomyocyte (Q35816653) (← links)
- Carbonic anhydrase IX is a predictive marker of doxorubicin resistance in early-stage breast cancer independent of HER2 and TOP2A amplification. (Q35835151) (← links)
- Doxorubicin increases intracellular hydrogen peroxide in PC3 prostate cancer cells (Q35962446) (← links)
- Dietary inorganic nitrate alleviates doxorubicin cardiotoxicity: mechanisms and implications. (Q35989515) (← links)
- Acute actions and novel targets of matrix metalloproteinases in the heart and vasculature (Q35995215) (← links)
- ErbB2 overexpression upregulates antioxidant enzymes, reduces basal levels of reactive oxygen species, and protects against doxorubicin cardiotoxicity. (Q36339400) (← links)
- Drug-induced mitochondrial dysfunction and cardiotoxicity (Q36339445) (← links)
- Simple quantitative detection of mitochondrial superoxide production in live cells (Q36431853) (← links)
- Oxidative stress, redox signaling, and metal chelation in anthracycline cardiotoxicity and pharmacological cardioprotection. (Q36568908) (← links)
- Iron chelators with topoisomerase-inhibitory activity and their anticancer applications (Q36568915) (← links)
- Sirt3 protects mitochondrial DNA damage and blocks the development of doxorubicin-induced cardiomyopathy in mice. (Q36902292) (← links)
- Doxorubicin induced heart failure: Phenotype and molecular mechanisms. (Q36912561) (← links)
- Liposomal daunorubicin as treatment for Kaposi's sarcoma (Q37004096) (← links)
- Role of superoxide, nitric oxide, and peroxynitrite in doxorubicin-induced cell death in vivo and in vitro (Q37200311) (← links)
- Poly-ADP ribose polymerase activates nuclear proteasome to degrade oxidatively damaged histones (Q37202186) (← links)
- Oxidative DNA base damage in MCF-10A breast epithelial cells at clinically achievable concentrations of doxorubicin (Q37219532) (← links)
- Development of a tumor-specific photoactivatable doxorubicin prodrug (Q37280632) (← links)
- The role of cell death and myofibrillar damage in contractile dysfunction of long-term cultured adult cardiomyocytes exposed to doxorubicin (Q37480989) (← links)
- Decreased Soluble Guanylate Cyclase Contributes to Cardiac Dysfunction Induced by Chronic Doxorubicin Treatment in Mice (Q37613120) (← links)
- BGP-15 Protects against Oxaliplatin-Induced Skeletal Myopathy and Mitochondrial Reactive Oxygen Species Production in Mice (Q37742823) (← links)
- Drug-induced oxidative stress and toxicity (Q38037517) (← links)
- Role of Drug Metabolism in the Cytotoxicity and Clinical Efficacy of Anthracyclines (Q38576762) (← links)
- Mechanisms of Doxorubicin Toxicity in Pancreatic β-Cells (Q38766953) (← links)
- Mitochondrial Function, Biology, and Role in Disease: A Scientific Statement From the American Heart Association. (Q38820532) (← links)
- Trastuzumab cardiotoxicity: from clinical trials to experimental studies (Q38974785) (← links)