Pages that link to "Q28218469"
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The following pages link to Amplification of PPM1D in human tumors abrogates p53 tumor-suppressor activity (Q28218469):
Displaying 50 items.
- PRAK is essential for ras-induced senescence and tumor suppression (Q24294592) (← links)
- The Wip1 Phosphatase acts as a gatekeeper in the p53-Mdm2 autoregulatory loop (Q24297633) (← links)
- Wild-type p53-induced phosphatase 1 dephosphorylates histone variant gamma-H2AX and suppresses DNA double strand break repair (Q24297983) (← links)
- DYRK2 is targeted to the nucleus and controls p53 via Ser46 phosphorylation in the apoptotic response to DNA damage (Q24299608) (← links)
- Protein kinase C delta regulates Ser46 phosphorylation of p53 tumor suppressor in the apoptotic response to DNA damage (Q24299870) (← links)
- Checkpoint recovery in cells: how a molecular understanding can help in the fight against cancer (Q24306163) (← links)
- PPM1D dephosphorylates Chk1 and p53 and abrogates cell cycle checkpoints (Q24527393) (← links)
- Oncogenic Wip1 phosphatase is inhibited by miR-16 in the DNA damage signaling pathway (Q24607221) (← links)
- p38alpha and p38gamma mediate oncogenic ras-induced senescence through differential mechanisms (Q24657888) (← links)
- Biological background of pediatric medulloblastoma and ependymoma: a review from a translational research perspective (Q24658389) (← links)
- The HBP1 transcriptional repressor participates in RAS-induced premature senescence (Q24672298) (← links)
- Novel hepatocellular carcinoma molecules with prognostic and therapeutic potentials (Q26824909) (← links)
- (Q28191114) (redirect page) (← links)
- Oncogenic properties of PPM1D located within a breast cancer amplification epicenter at 17q23 (Q28218453) (← links)
- Wip-ing out cancer (Q28253700) (← links)
- DNA damage and the balance between survival and death in cancer biology (Q28271163) (← links)
- Regulation of the antioncogenic Chk2 kinase by the oncogenic Wip1 phosphatase (Q28283888) (← links)
- Role of mitogen-activated protein kinase kinase 4 in cancer (Q28301942) (← links)
- Mosaic PPM1D mutations are associated with predisposition to breast and ovarian cancer (Q28388006) (← links)
- The effects of chromium(VI) on the thioredoxin system: implications for redox regulation (Q28397704) (← links)
- Wip1 suppresses ovarian cancer metastasis through the ATM/AKT/Snail mediated signaling (Q28828189) (← links)
- Wee1 inhibition potentiates Wip1-dependent p53-negative tumor cell death during chemotherapy (Q28833606) (← links)
- Live or let die: the cell's response to p53 (Q29547663) (← links)
- MAP kinase signalling pathways in cancer (Q29615542) (← links)
- Anticancer effect of Tamarix gallica extracts on human colon cancer cells involves Erk1/2 and p38 action on G2/M cell cycle arrest (Q30390159) (← links)
- Dysregulated estrogen receptor signaling in the hypothalamic-pituitary-ovarian axis leads to ovarian epithelial tumorigenesis in mice (Q30414345) (← links)
- A high-throughput assay for phosphoprotein-specific phosphatase activity in cellular extracts (Q30414621) (← links)
- A chemical inhibitor of PPM1D that selectively kills cells overexpressing PPM1D. (Q33294239) (← links)
- Retinoblastoma loss modulates DNA damage response favoring tumor progression (Q33382723) (← links)
- The oncogenic phosphatase WIP1 negatively regulates nucleotide excision repair (Q33572384) (← links)
- Excitability in the p53 network mediates robust signaling with tunable activation thresholds in single cells (Q33572789) (← links)
- Progesterone action in human tissues: regulation by progesterone receptor (PR) isoform expression, nuclear positioning and coregulator expression (Q33595229) (← links)
- Nuclear factor-kappaB (NF-kappaB) is a novel positive transcriptional regulator of the oncogenic Wip1 phosphatase (Q33648301) (← links)
- Phosphatases reverse p53-mediated cell cycle checkpoints (Q33665426) (← links)
- The mitogen-activated protein (MAP) kinases p38 and extracellular signal-regulated kinase (ERK) are involved in hepatocyte-mediated phenotypic switching in prostate cancer cells (Q33676246) (← links)
- Transcriptional response of lymphoblastoid cells to ionizing radiation (Q33679034) (← links)
- Inactivation of p38 MAPK contributes to stem cell-like properties of non-small cell lung cancer (Q33688798) (← links)
- WIP1 phosphatase as pharmacological target in cancer therapy. (Q33720757) (← links)
- Expression of a homeostatic regulator, Wip1 (wild-type p53-induced phosphatase), is temporally induced by c-Jun and p53 in response to UV irradiation (Q33726581) (← links)
- Exome sequencing identifies somatic gain-of-function PPM1D mutations in brainstem gliomas (Q33813999) (← links)
- DOTS-Finder: a comprehensive tool for assessing driver genes in cancer genomes. (Q33858472) (← links)
- Stress signaling and the shaping of the mammary tissue in development and cancer (Q33896062) (← links)
- Posttranslational modification of p53: cooperative integrators of function (Q33899450) (← links)
- Abnormality of pl6/p38MAPK/p53/Wipl pathway in papillary thyroid cancer (Q33972927) (← links)
- Wip1 directly dephosphorylates gamma-H2AX and attenuates the DNA damage response (Q33994571) (← links)
- p38α MAPK pathway: a key factor in colorectal cancer therapy and chemoresistance. (Q34008781) (← links)
- Progenitor-derived hepatocellular carcinoma model in the rat. (Q34037698) (← links)
- RNA interference (RNAi) screening approach identifies agents that enhance paclitaxel activity in breast cancer cells. (Q34048144) (← links)
- PPM1D silencing by RNA interference inhibits the proliferation of lung cancer cells (Q34136105) (← links)
- Phosphatases in the cellular response to DNA damage. (Q34198570) (← links)